Multi-undulant T-U-wave, sinus bradycardia and long QT syndrome: A possible phenotype of mutant genes controlling the inward potassium rectifiers

Acta Paediatrica Sinica

Volumn 38, Issue 4, 1997, Pages 267-275

  Shen, Ching Tsuen ^a  

^a CATHAY GENERAL HOSPITAL   (Taiwan)

Author keywords

Congenital long QT syndrome; HERG; Holter recordings; KvLQT1

Indexed keywords

POTASSIUM CHANNEL;

ADOLESCENT; ADULT; ARTICLE; CHILD; CHROMOSOME 11P; CHROMOSOME 7Q; CLINICAL ARTICLE; CLINICAL FEATURE; CONTROLLED STUDY; FEMALE; GENE MUTATION; GENETIC LINKAGE; HEART REPOLARIZATION; HOLTER MONITORING; HUMAN; INFANT; LONG QT SYNDROME; MALE; POTASSIUM CURRENT; PRESCHOOL CHILD; RR INTERVAL; SCHOOL CHILD; SINUS BRADYCARDIA;

ADOLESCENT; BRADYCARDIA; CATION TRANSPORT PROTEINS; CHILD; CHILD, PRESCHOOL; DNA-BINDING PROTEINS; ELECTROCARDIOGRAPHY; ETHER-A-GO-GO POTASSIUM CHANNELS; FEMALE; HUMANS; INFANT; LONG QT SYNDROME; MALE; MUTATION; PHENOTYPE; POTASSIUM CHANNELS; POTASSIUM CHANNELS, VOLTAGE-GATED; TRANS-ACTIVATORS;

EID: 0030879024     PISSN: 00016578     EISSN: None     Source Type: Journal    
DOI: None     Document Type: Article

References (2)

1. Boyden PA. Cellular electrophysiologic basis of cardiac arrhythmias. Am J Cardiol 1996; 78(suppl 4A):4-11.
2. Deal KK, England SK, Kamkun MM. Molecular physiology of cardiac potassium channels. Phyiol Rev 1996; 76:49-67.