Induction of apoptosis by overexpression of the PU. 1/Spi-1 gene in murine erythroleukemia cells

Leukemia

Volumn 11, Issue SUPPL. 3, 1997, Pages 480-481

  Yamada, Toshiyuki ^a   Oikawa, Tsuneyuki ^a  

^a SASAKI INSTITUTE   (Japan)

Author keywords

[No Author keywords available]

Indexed keywords

BETA GLOBIN; CELL DNA; MYC PROTEIN; PROTEIN BCL 2; PROTEIN P53; TRANSCRIPTION FACTOR SP1;

ANIMAL CELL; APOPTOSIS; CELL DIFFERENTIATION; CONFERENCE PAPER; CONTROLLED STUDY; DNA SEQUENCE; ERYTHROLEUKEMIA; FRIEND LEUKEMIA VIRUS; GENE ACTIVATION; GENE EXPRESSION; LEUKEMOGENESIS; MALIGNANT TRANSFORMATION; MOUSE; NONHUMAN; ONCOGENE; PLASMID; PRIORITY JOURNAL; SPLEEN FOCUS FORMING VIRUS;

EID: 0030873516     PISSN: 08876924     EISSN: None     Source Type: Journal    
DOI: None     Document Type: Article

References (5)

1. Moreau-Gachelin F. et al. Spi-1 is a putative oncogene in virally induced murine erythroleukemias. Nature, 1988; 331: 277-280.
2. Paul R. et al. A common site for immortalizing proviral integrations in Friend erythroleukemia: molecular cloning and characterization. J. Virol., 1989; 63: 4958-4961.
3. Klemsz M. et al. The macrophage and B cell-specific transcription factor PU. 1 is related to the ets oncogene. Cell, 1990; 61: 113-124.
4. Moreau-Gachelin F. et al. The PU. 1 transcription factor is the product of the putative oncogene Spi-1. Cell, 1990; 61: 1165-1166.
5. Scott E. et al. Requirement of transcription factor PU. 1 in the development of multiple hematopoietic lineages. Science, 1994; 265: 1573-1577.