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Cloning of human mineralocorticoid receptor complementary DNA: Structural and functional kinship with the glucocorticoid receptors
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Affiza, J.L., Weinberger, C., Glaser, T.M., Handelin, B.L., Housman, D.E., Evans, R.M. Cloning of human mineralocorticoid receptor complementary DNA: structural and functional kinship with the glucocorticoid receptors. Science 1987; 237: 268-275.
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The nuclear receptor superfamily: The second decade
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Mangelsdorf, D.J., Thummel, C., Beato, M., Herrlich, P., Schutz, G., Umesono, K., Blumberg, B., Kastner, P., Mark, M., Chambon, P., Evans, R.M. The nuclear receptor superfamily: the second decade. Cell 1995; 83: 835-839.
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Myles, K., Funder, J.W. Type I (mineralocorticoid) receptors in the guinea pig. Am J Physiol 1994; 267: E268-E272.
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Naray-Fejes-Toth, A., Fejes-Toth, G. Glucocorticoid receptors mediate mineralocorticoid-like effects in cultured collecting duct cells. Am J Physiol. 1990; 259: F672-F678.
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Funder, J.W., Pearce, P., Myles, K., Roy, L.P. Apparent mineralocorticoid excess, pseudohypoaldosteronism and urinary electrolyte excretion: towards a redefinition of "mineralocorticoid" action. FASEB J 1990; 4: 3234-3238.
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Mutations in the 11β hydroxysteroid dehydrogenase Type 2 enzyme associated with hypertension and possibly stillbirth
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Krozowski, Z.S., Stewart, P., Obeyesekere, V., Li, K., Feffari, P. Mutations in the 11β hydroxysteroid dehydrogenase Type 2 enzyme associated with hypertension and possibly stillbirth. Clin Exp Hypertens.(this issue).
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A mutation in the HSD11B2 gene in a family with Apparent Mineralocorticoid Excess
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Wilson, R.C., Krozowski, Z.S., Li, K., Obeyesekere, V., Razzaghy-Azar, M.M., Harbison, D., Wei, J.Q., Shackleton, C.H.L., Funder, J.W., New, M.I. A mutation in the HSD11B2 gene in a family with Apparent Mineralocorticoid Excess. J Clin Endocrinol Metab 1995; 80: 2263-2266.
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10
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Human hypertension caused by mutations in the gene encoding the kidney isozyme of 110-hydroxysteroid dehydrogenase
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Mune, T., Rogerson, F.M., Nikkila, H., Agarwal, A.K., White, P.C. Human hypertension caused by mutations in the gene encoding the kidney isozyme of 110-hydroxysteroid dehydrogenase. Nat Genet 1995; 10: 394-399.
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Several homozygous mutations in the gene for 11β-hydroxysteroid dehydrogenase Type 2 in patients with Apparent Mineralocorticoid Excess
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Wilson, R.C., Harbison, M.D., Krozowski, Z.S., Funder, J.W., Shackleton, C.H.L., Hanauske-Abel, H.M., Wei, J-Q., Hertecant, J., Moran, A., Neiberger, R.E., Balfe, J.W., Fattach, A., Daneman, D., Licholai, T., New, M.I. Several homozygous mutations in the gene for 11β-hydroxysteroid dehydrogenase Type 2 in patients with Apparent Mineralocorticoid Excess. J Clin Endocrinol Metab 1995; 80: 3145-3150.
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Albiston, A., Obeyesekere, V., Smith, R., Krozowski, Z.S. Cloning and tissue distribution of the human 11-β hydroxysteroid dehydrogenase type II enzyme. Mol Cell Endocrinol 1994; 105: R11-R17.
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Exclusion of cortic oster one from epithelial mineralocorticoid receptors is insufficient for selectivity of aldosterone action: In vivo binding studies
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Funder, J.W., Myles, K. Exclusion of cortic oster one from epithelial mineralocorticoid receptors is insufficient for selectivity of aldosterone action: in vivo binding studies. Endocrinology 1996, in press.
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Gomez-Sanchez, E.P., Venkataraman, M.T., Thwaites, D., Fort, C. ICV infusion of corticosterone antagonizes ICV-aldosterone hypertension. Am J Physiol 1990; 258: E649-E653.
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Determinants of cardiac fibrosis in experimental hypermineralocorticoid states
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Young, M., Head, G., Funder, J.W. Determinants of cardiac fibrosis in experimental hypermineralocorticoid states. Am J Physiol 1995; 269: E657-E662.
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Young M., Fullerton, M., Dilley, R., Funder, J.W. Mineralocorticoids, hypertension and cardiac fibrosis. J Clin Invest 1994; 93: 2578-2583.
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Guarda, E., Myers, P., Brilla, C., Tyagi, S., Weber, K.T. Endothelial cell induced modulation of cardiac fibroblast collagen metabolism. Cardiovasc Res. 1993; 27: 1004-1008.
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0029762716
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High glucose stimulates aldosterone-induced hypertrophy via Type I mineralocorticoid receptors in neonatal rat cardiomyocytes
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Sato, A., Funder, J.W. High glucose stimulates aldosterone-induced hypertrophy via Type I mineralocorticoid receptors in neonatal rat cardiomyocytes. Endocrinology 1996, in press.
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Role of glucocorticoid in the development of glycyrrhizin-induced hypertension
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