Studies of human, mouse and yeast homologues indicate a mitochondrial function for frataxin

Nature Genetics

Volumn 16, Issue 4, 1997, Pages 345-351

  Koutnikova, Hana ^a   Campuzano, Victoria ^a   Foury, Françoise ^b   Dollé, Pascal ^a   Cazzalini, Ornella ^b   Koenig, Michel ^a  

^a INSERM   (France)

^b UNIVERSITÉ CATHOLIQUE DE LOUVAIN   (Belgium)

Author keywords

[No Author keywords available]

Indexed keywords

FRATAXIN; PROTEIN; UNCLASSIFIED DRUG;

AMINO ACID SEQUENCE; ARTICLE; AUTOSOMAL RECESSIVE DISORDER; DEGENERATIVE DISEASE; FRIEDREICH ATAXIA; HUMAN; IN SITU HYBRIDIZATION; MITOCHONDRION; MOUSE; NONHUMAN; PRIORITY JOURNAL; PROTEIN ANALYSIS; PROTEIN EXPRESSION; SACCHAROMYCES CEREVISIAE; SEQUENCE HOMOLOGY;

AMINO ACID SEQUENCE; ANIMALS; ANIMALS, NEWBORN; EMBRYO; GENE DELETION; GENE EXPRESSION; HELA CELLS; HUMANS; IRON-BINDING PROTEINS; MICE; MITOCHONDRIA; MOLECULAR SEQUENCE DATA; PHENOTYPE; PHOSPHOTRANSFERASES (ALCOHOL GROUP ACCEPTOR); RNA, MESSENGER; SACCHAROMYCES CEREVISIAE;

ATAXIA; SACCHAROMYCES CEREVISIAE;

EID: 0030813487     PISSN: 10614036     EISSN: None     Source Type: Journal    
DOI: 10.1038/ng0897-345     Document Type: Article

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