Aggregation and binding substances enhance pathogenicity in a rabbit model of Enterococcus faecalis endocarditis

Advances in Experimental Medicine and Biology

Volumn 418, Issue , 1997, Pages 789-791

  Schlievert, P M ^a   Dunny, G M ^a   Stoehr, J A ^b   Assimacopoulos, A P ^b  

^a UNIVERSITY OF MINNESOTA MEDICAL SCHOOL   (United States)

^b St Paul Infectious Disease Associates   (United States)

Author keywords

[No Author keywords available]

Indexed keywords

ANIMAL MODEL; BACTERIAL ENDOCARDITIS; CELL AGGREGATION; CONFERENCE PAPER; ENTEROCOCCUS FAECALIS; INFECTION RISK; NONHUMAN; PATHOGENICITY; PRIORITY JOURNAL; RABBIT;

EID: 0030763342     PISSN: 00652598     EISSN: None     Source Type: Book Series    
DOI: 10.1007/978-1-4899-1825-3_186     Document Type: Conference Paper

References (2)

1. Chow JW, Thal LA, Perri MB, Vazquez JA, Donabedian SM, Clewell DB, Zervos MJ. Plasmid-associated hemolysin and aggregation substance production contribute to virulence in eperimental enterococcal endocarditis. Antimicrob Agent Chemother 1993;37:2474-2477.
2. Olmsted SB, Kao SM, van Putte LJ, Gallo JC, Dunny GM. Role of pheromone-inducible surface protein Asc10 in mating aggregate formation and conjugal transfer of the Enterococcus faecalis plasmid pCF10. J Bacteriol 1991;173:7665-7672.