Mnt: A novel Max-interacting protein and Myc antagonist

Current Topics in Microbiology and Immunology

Volumn 224, Issue , 1997, Pages 115-121

  Hurlin, P J ^a   Queva C ^a   Eisenman, R N ^a  

^a Fred Hutchinson Cancer Research Center   (United States)

Author keywords

[No Author keywords available]

Indexed keywords

MESSENGER RNA; MYC PROTEIN; REPRESSOR PROTEIN;

ANIMAL CELL; ANIMAL TISSUE; CELL DIFFERENTIATION; CELL PROLIFERATION; CELL TRANSFORMATION; CONFERENCE PAPER; CONTROLLED STUDY; EMBRYONAL CARCINOMA; GENE ACTIVATION; GENE EXPRESSION REGULATION; MOUSE; NONHUMAN; ONCOGENE C MYC; PRIORITY JOURNAL; PROMOTER REGION; PROTEIN DOMAIN; PROTEIN PROTEIN INTERACTION; REPORTER GENE; REPRESSOR GENE; TRANSCRIPTION REGULATION;

ANIMALIA;

EID: 0030756409     PISSN: 0070217X     EISSN: None     Source Type: Book Series    
DOI: 10.1007/978-3-642-60801-8_11     Document Type: Conference Paper

References (5)

1. Amin, C., A.J. Wagner and N.Hay. 1993. Sequence-specific transcriptional activation by Myc and repression by Max. Mol. Cell. Biol. 13: 383-390.
2. Ayer, D.E., L. Kretzner and R.N. Eisenman. 1993a. Mad: A heterodimeric partner for Max that antagonizes Myc transcriptional activity. Cell 72: 211-222.
3. Ayer, D.E. and R.N. Eisenman. 1993b. A switch from Myc:Max to Mad:Max heterocomplexes accompanies monocyte/macrophage differentiation. Genes & Dev. 7: 2110-2119.
4. Ayer, D.E., Q. A. Lawrence and R.N. Eisenman. 1995. Mad-Max transcriptional repression is mediated by ternary complex formation with mammalian homologs of the yeast represser Sin3. Cell 80: 767-776.
5. Dlackwell, T.K., L. Kretzner, E.M. Blackwood, R.N. Eisenman and H. Weintraub. 1990. Sequence-specific DNA-binding by the c-Myc protein. Science 250: 1149-1151.