HOP-1, a Caenorhabditis elegans presenilin, appears to be functionally redundant with SEL-12 presenilin and to facilitate LIN-12 and GLP-1 signaling

Proceedings of the National Academy of Sciences of the United States of America

Volumn 94, Issue 22, 1997, Pages 12204-12209

  Li, Xiajun ^a   Greenwald, Iva ^a,b  

^a College of Physicians and Surgeons ^*  (United States)

^b Dept Biochem and Molec Biophys ^*  (United States)

Author keywords

[No Author keywords available]

Indexed keywords

COMPLEMENTARY DNA; MEMBRANE PROTEIN; PRESENILIN; RNA; UNCLASSIFIED DRUG;

ALZHEIMER DISEASE; AMINO ACID SEQUENCE; ARTICLE; CAENORHABDITIS ELEGANS; DEGENERATIVE DISEASE; GENE LOCUS; GENE MUTATION; NONHUMAN; PHENOTYPE; PRIORITY JOURNAL; PROTEIN ANALYSIS; TRANSGENIC ANIMAL;

ALZHEIMER DISEASE; AMINO ACID SEQUENCE; ANIMALS; ANIMALS, GENETICALLY MODIFIED; CAENORHABDITIS ELEGANS; CAENORHABDITIS ELEGANS PROTEINS; CLONING, MOLECULAR; DNA, COMPLEMENTARY; FEMALE; GENES, LETHAL; GENETIC COMPLEMENTATION TEST; HELMINTH PROTEINS; HUMANS; MEMBRANE GLYCOPROTEINS; MEMBRANE PROTEINS; MOLECULAR SEQUENCE DATA; OVIPOSITION; PHENOTYPE; PROTEIN CONFORMATION; SEQUENCE ANALYSIS, DNA; SEQUENCE HOMOLOGY, AMINO ACID; SIGNAL TRANSDUCTION; SPECIES SPECIFICITY;

ANIMALIA; CAENORHABDITIS ELEGANS; HUMULUS; MAMMALIA;

EID: 0030671560     PISSN: 00278424     EISSN: None     Source Type: Journal    
DOI: 10.1073/pnas.94.22.12204     Document Type: Article

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