-
1
-
-
0029026548
-
FADD, a novel death domain-containing protein, interacts with the death domain of Fas and initiates apoptosis
-
Chinnaiyan, A.M., O'Rourke, K., Tewari, M. & Dixit, V.M. (1995) FADD, a novel death domain-containing protein, interacts with the death domain of Fas and initiates apoptosis. Cell, 81, 505-512.
-
(1995)
Cell
, vol.81
, pp. 505-512
-
-
Chinnaiyan, A.M.1
O'Rourke, K.2
Tewari, M.3
Dixit, V.M.4
-
2
-
-
0029965280
-
FADD/MORT1 is a common mediator of CD95 (Fas/APO-1) and tumour necrosis factor receptor-induced apoptosis
-
Chinnaiyan, A.M., Tepper, C.G., Seldin, M.F., O'Rourke, K., Kischkel, F.C., Hellbardt, S., Krammer, P.H., Peter, M.E. & Dixit, V.M. (1996) FADD/MORT1 is a common mediator of CD95 (Fas/APO-1) and tumour necrosis factor receptor-induced apoptosis. Journal of Biological Chemistry, 271, 4961-4965.
-
(1996)
Journal of Biological Chemistry
, vol.271
, pp. 4961-4965
-
-
Chinnaiyan, A.M.1
Tepper, C.G.2
Seldin, M.F.3
O'Rourke, K.4
Kischkel, F.C.5
Hellbardt, S.6
Krammer, P.H.7
Peter, M.E.8
Dixit, V.M.9
-
3
-
-
0028095477
-
Apoptotic signaling through CD95 (Fas/APO-1) activates an acidic sphingomyelinase
-
Cifone, M.G., De Maria, R., Roncaioli, P., Rippo, M.R., Azuma, M., Lanier, L.L., Santoni, A. & Testi, R. (1994) Apoptotic signaling through CD95 (Fas/APO-1) activates an acidic sphingomyelinase. Journal of Experimental Medicine, 180, 1547-1552.
-
(1994)
Journal of Experimental Medicine
, vol.180
, pp. 1547-1552
-
-
Cifone, M.G.1
De Maria, R.2
Roncaioli, P.3
Rippo, M.R.4
Azuma, M.5
Lanier, L.L.6
Santoni, A.7
Testi, R.8
-
4
-
-
0029066697
-
Contenders in FasL/TNF death signaling
-
Cleveland, J.L. & Ihle, J.N. (1995) Contenders in FasL/TNF death signaling. Cell, 81, 479-482.
-
(1995)
Cell
, vol.81
, pp. 479-482
-
-
Cleveland, J.L.1
Ihle, J.N.2
-
5
-
-
0029978182
-
Sequential activation of ICE-like and CPP32-like proteases during Fas-mediated apoptosis
-
Enari, M., Talanian, R.V., Wong, W.W. & Nagata, S. (1996) Sequential activation of ICE-like and CPP32-like proteases during Fas-mediated apoptosis. Nature, 380, 723-726.
-
(1996)
Nature
, vol.380
, pp. 723-726
-
-
Enari, M.1
Talanian, R.V.2
Wong, W.W.3
Nagata, S.4
-
6
-
-
0029025441
-
Dominant interfering Fas gene mutations impair apoptosis in a human autoimmune lymphoproliferative syndrome
-
Fisher, G.H., Rosenberg, F.J., Straus, S.E., Dale, J.K., Middelton, L.A., Lin, A.Y., Strober, W., Lenardo, M.J. & Puck, J.M. (1995) Dominant interfering Fas gene mutations impair apoptosis in a human autoimmune lymphoproliferative syndrome. Cell, 81, 935-946.
-
(1995)
Cell
, vol.81
, pp. 935-946
-
-
Fisher, G.H.1
Rosenberg, F.J.2
Straus, S.E.3
Dale, J.K.4
Middelton, L.A.5
Lin, A.Y.6
Strober, W.7
Lenardo, M.J.8
Puck, J.M.9
-
7
-
-
0029949257
-
TNF dependent recruitment of the protein kinase RIP to the TNF receptor-1 signaling complex
-
Hsu, H., Huang, J., Shu, H.,-B., Baichwal, V. & Goeddel, D.V. (1996) TNF dependent recruitment of the protein kinase RIP to the TNF receptor-1 signaling complex. Immunity, 4, 387-396.
-
(1996)
Immunity
, vol.4
, pp. 387-396
-
-
Hsu, H.1
Huang, J.2
Shu, H.-B.3
Baichwal, V.4
Goeddel, D.V.5
-
8
-
-
0029007855
-
The TNF receptor 1-associated protein TRADD signals cell death and NF-κB activation
-
Hsu, H., Xiong, J. & Goeddel, D.V. (1995) The TNF receptor 1-associated protein TRADD signals cell death and NF-κB activation. Cell, 81, 495-504.
-
(1995)
Cell
, vol.81
, pp. 495-504
-
-
Hsu, H.1
Xiong, J.2
Goeddel, D.V.3
-
9
-
-
0027161158
-
Effect of bcl-2 on Fas antigen-mediated cell death
-
Itoh, N., Tsujimoto, Y. & Nagata, S. (1993) Effect of bcl-2 on Fas antigen-mediated cell death. Journal of Immunology, 151, 621-627.
-
(1993)
Journal of Immunology
, vol.151
, pp. 621-627
-
-
Itoh, N.1
Tsujimoto, Y.2
Nagata, S.3
-
10
-
-
0025916387
-
The polypeptide encoded by the cDNA for human cell surface antigen Fas can mediate apoptosis
-
Itoh, N., Yonehara, S., Ishii, A., Yonehara, M., Mizushima, S.-I., Sameshima, M., Hase, A., Seto, Y. & Nagata, S. (1991) The polypeptide encoded by the cDNA for human cell surface antigen Fas can mediate apoptosis. Cell, 66, 233-243.
-
(1991)
Cell
, vol.66
, pp. 233-243
-
-
Itoh, N.1
Yonehara, S.2
Ishii, A.3
Yonehara, M.4
Mizushima, S.-I.5
Sameshima, M.6
Hase, A.7
Seto, Y.8
Nagata, S.9
-
11
-
-
0028110928
-
Fas and perform pathways as major mechanisms of T-cell-mediated cytotoxicity
-
Kagi, D., Vignaux, F., Ledermann, B., Burki, K., Depraetere, V., Nagata, S., Hengartner, H. & Golstein, P. (1994) Fas and perform pathways as major mechanisms of T-cell-mediated cytotoxicity. Nature, 265, 528-530.
-
(1994)
Nature
, vol.265
, pp. 528-530
-
-
Kagi, D.1
Vignaux, F.2
Ledermann, B.3
Burki, K.4
Depraetere, V.5
Nagata, S.6
Hengartner, H.7
Golstein, P.8
-
12
-
-
0028885701
-
Fas receptor (CD95)-mediated apoptosis is induced in leukemic cells entering G1B compartment of the cell cycle
-
Komada, Y., Zhou, Y.-W., Zhang, X.-L., Xue, H.-L. Sakai, H., Tanaka, S., Sakatoku, H. & Sakurai, M. (1995) Fas receptor (CD95)-mediated apoptosis is induced in leukemic cells entering G1B compartment of the cell cycle. Blood, 86, 3848-3860.
-
(1995)
Blood
, vol.86
, pp. 3848-3860
-
-
Komada, Y.1
Zhou, Y.-W.2
Zhang, X.-L.3
Xue, H.-L.4
Sakai, H.5
Tanaka, S.6
Sakatoku, H.7
Sakurai, M.8
-
13
-
-
15844412409
-
FLICE, a novel FADD-homologous ICE/CED-3-like protease, is recruited to the CD95 (Fas/APO-1) death-inducing signaling complex
-
Muzio, M., Chinnaiyan, A.M., Kischkel, F.C., O'Rourke, K., Shevchenko, A., Ni, J., Scaffidi, C., Bretz, J.D., Zhang, M., Gentz, R., Mann, M., Krammer, P.H., Peter, M.E. & Dixit, V.M. (1996) FLICE, a novel FADD-homologous ICE/CED-3-like protease, is recruited to the CD95 (Fas/APO-1) death-inducing signaling complex. Cell, 85, 817-827.
-
(1996)
Cell
, vol.85
, pp. 817-827
-
-
Muzio, M.1
Chinnaiyan, A.M.2
Kischkel, F.C.3
O'Rourke, K.4
Shevchenko, A.5
Ni, J.6
Scaffidi, C.7
Bretz, J.D.8
Zhang, M.9
Gentz, R.10
Mann, M.11
Krammer, P.H.12
Peter, M.E.13
Dixit, V.M.14
-
14
-
-
0026706261
-
Purification and molecular cloning of the APO-1 cell surface antigen, a member of the TNF/ NGF receptor superfamily
-
Oehm, A., Behrmann, I., Falk, W., Li-Weber, M., Maier, G., Klas, C., Richards, S., Dhein, J., Daniel, P.T., Knipping, E., Trauth, B.C., Ponstingl, H. & Krammer, P.H. (1992) Purification and molecular cloning of the APO-1 cell surface antigen, a member of the TNF/ NGF receptor superfamily. Journal of Biological Chemistry, 15, 10709-10715.
-
(1992)
Journal of Biological Chemistry
, vol.15
, pp. 10709-10715
-
-
Oehm, A.1
Behrmann, I.2
Falk, W.3
Li-Weber, M.4
Maier, G.5
Klas, C.6
Richards, S.7
Dhein, J.8
Daniel, P.T.9
Knipping, E.10
Trauth, B.C.11
Ponstingl, H.12
Krammer, P.H.13
-
15
-
-
0027402369
-
Fas involvement in Ca2+-dependent T cell-mediated cytotoxicity
-
Rouvier, E., Luciani, M.-F. & Golstein, P. (1993) Fas involvement in Ca2+-dependent T cell-mediated cytotoxicity. Journal of Expermental Medicine, 177, 195-200.
-
(1993)
Journal of Expermental Medicine
, vol.177
, pp. 195-200
-
-
Rouvier, E.1
Luciani, M.-F.2
Golstein, P.3
-
16
-
-
0029066512
-
FAP-1: A protein tyrosine phosphatase that associates with Fas
-
Sato, T. Irie, S., Kitada, S. & Reed, J.C. (1995) FAP-1: a protein tyrosine phosphatase that associates with Fas. Science, 268, 411-415.
-
(1995)
Science
, vol.268
, pp. 411-415
-
-
Sato, T.1
Irie, S.2
Kitada, S.3
Reed, J.C.4
-
17
-
-
0029054725
-
RIP: A novel protein containing a death domain that interacts with Fas/ APO-1 (CD95) in yeast and causes cell death
-
Stanger, B.Z., Leder, P., Lee, T.-H., Kim, E. & Seed, B. (1995) RIP: a novel protein containing a death domain that interacts with Fas/ APO-1 (CD95) in yeast and causes cell death. Cell, 81, 513-523.
-
(1995)
Cell
, vol.81
, pp. 513-523
-
-
Stanger, B.Z.1
Leder, P.2
Lee, T.-H.3
Kim, E.4
Seed, B.5
-
18
-
-
0028053599
-
Human Fas ligand: Gene structure, chromosomal location and species specificity
-
Takahashi, T., Tanaka, M., Inazawa, J., Abe, T., Suda, T. & Nagata, S. (1994) Human Fas ligand: gene structure, chromosomal location and species specificity. International Immunology, 6, 1567-1574.
-
(1994)
International Immunology
, vol.6
, pp. 1567-1574
-
-
Takahashi, T.1
Tanaka, M.2
Inazawa, J.3
Abe, T.4
Suda, T.5
Nagata, S.6
-
19
-
-
0028873964
-
Fas-and tumour necrosis factor-induced apoptosis is inhibited by the poxvirus CrmA gene product
-
Tewari, M. & Dixit, V.M. (1995) Fas-and tumour necrosis factor-induced apoptosis is inhibited by the poxvirus CrmA gene product. Journal of Biological Chemistry, 270, 3255-3260.
-
(1995)
Journal of Biological Chemistry
, vol.270
, pp. 3255-3260
-
-
Tewari, M.1
Dixit, V.M.2
-
20
-
-
0028926523
-
TCR/CD3 coupling to Fas-based cytotoxicity
-
Vignaux, F., Vivier E., Malissen, B., Depraetere, V., Nagata, S. & Golstein, P. (1995) TCR/CD3 coupling to Fas-based cytotoxicity. Journal of Experimental Medicine, 181, 781-786.
-
(1995)
Journal of Experimental Medicine
, vol.181
, pp. 781-786
-
-
Vignaux, F.1
Vivier, E.2
Malissen, B.3
Depraetere, V.4
Nagata, S.5
Golstein, P.6
-
21
-
-
0026568919
-
Lymphoproliferation disorder in mice explained by defects in Fas antigen that mediates apoptosis
-
Watanabe-Fukunaga, R., Brannan, C.I., Copeland, N.G., Jenkins, N.A. & Nagata, S. (1992) Lymphoproliferation disorder in mice explained by defects in Fas antigen that mediates apoptosis. Nature, 356, 314-318.
-
(1992)
Nature
, vol.356
, pp. 314-318
-
-
Watanabe-Fukunaga, R.1
Brannan, C.I.2
Copeland, N.G.3
Jenkins, N.A.4
Nagata, S.5
-
22
-
-
0028108015
-
Functional dichotomy of neural and acidic sphingomyelinases in tumour necrosis factor signaling
-
Wiegmann, K., Schutze, S., Machleidt, T., Witte, D. & Kronke, M. (1994) Functional dichotomy of neural and acidic sphingomyelinases in tumour necrosis factor signaling. Cell, 78, 1005-1105.
-
(1994)
Cell
, vol.78
, pp. 1005-1105
-
-
Wiegmann, K.1
Schutze, S.2
Machleidt, T.3
Witte, D.4
Kronke, M.5
-
23
-
-
0024596722
-
Cell-killing monoclonal antibody (anti-Fas) to a cell surface antigen codownregulated with the receptor of tumour necrosis factor
-
Yonehara, S., Ishii, A. & Yonehara, M.A. (1989) Cell-killing monoclonal antibody (anti-Fas) to a cell surface antigen codownregulated with the receptor of tumour necrosis factor. Journal of Experimental Medicine, 169, 1747-1756.
-
(1989)
Journal of Experimental Medicine
, vol.169
, pp. 1747-1756
-
-
Yonehara, S.1
Ishii, A.2
Yonehara, M.A.3
|