Retroviral transfer of acid α-glucosidase cDNA to enzyme-deficient myoblasts results in phenotypic spread of the genotypic correction by both secretion and fusion

Human Gene Therapy

Volumn 8, Issue 13, 1997, Pages 1555-1563

  Zaretsky, Joseph Z ^a  

^a NATIONAL INSTITUTE OF ARTHRITIS AND MUSCULOSKELETAL AND SKIN DISEASES   (United States)

Author keywords

[No Author keywords available]

Indexed keywords

COMPLEMENTARY DNA; GLUCAN 1,4 ALPHA GLUCOSIDASE; GLYCOGEN; SOMATOMEDIN B RECEPTOR;

ARTICLE; CELL FUSION; CONTROLLED STUDY; ENDOCYTOSIS; ENZYME ACTIVITY; ENZYME RELEASE; EXPRESSION VECTOR; GENE EXPRESSION; GENE TRANSFER; GENETIC TRANSDUCTION; GENOTYPE; GLYCOGEN STORAGE DISEASE TYPE 2; HUMAN; HUMAN CELL; HUMAN TISSUE; LYSOSOME; MUSCLE DEVELOPMENT; MYOBLAST; MYOTUBE; PHENOTYPE; RETROVIRUS;

UNIDENTIFIED RETROVIRUS;

EID: 0030669115     PISSN: 10430342     EISSN: None     Source Type: Journal    
DOI: 10.1089/hum.1997.8.13-1555     Document Type: Article

References (1)

1. BARR, E., and LEIDEN, J.M. (1991). Systemic delivery of recombinant proteins by genetically modified myoblasts. Science 254, 1507-1509.