β-amyloid-induced cerebrovascular endothelial dysfunction

Annals of the New York Academy of Sciences

Volumn 826, Issue , 1997, Pages 447-451

  Thomas, Tom ^a,b   McLendon, Chris ^a   Sutton, E Truitt ^a   Thomas, George ^a  

^a UNIVERSITY OF SOUTH FLORIDA   (United States)

^b Woodlands Med and Research Center   (United States)

Author keywords

[No Author keywords available]

Indexed keywords

AMYLOID BETA PROTEIN; BRADYKININ; OXYGEN RADICAL; REACTIVE OXYGEN METABOLITE; SEROTONIN; SUPEROXIDE DISMUTASE;

ALZHEIMER DISEASE; ANIMAL TISSUE; BRAIN ARTERY; CEREBROVASCULAR DISEASE; CONFERENCE PAPER; CONTROLLED STUDY; ELECTRON MICROSCOPY; ENDOTHELIUM LESION; NONHUMAN; SENILE PLAQUE; VASCULAR ENDOTHELIUM; VASODILATATION;

ANIMALIA;

EID: 0030668818     PISSN: 00778923     EISSN: None     Source Type: Book Series    
DOI: 10.1111/j.1749-6632.1997.tb48501.x     Document Type: Conference Paper

References (5)

1. KALARIA, R. N. 1993. Cerebral microvasculature and immunological factors in Alzheimer's Disease Clin. Neurosci. 1: 204-211.
2. THOMAS, T. et al. 1996. β-amyloid-mediated vasoactivity and vascular endothelial damage. Nature 380: 168-171.
3. MAYHAN, W. G. et al. 1990. Effects of aging on response of cerebral arterioles. Am. J. Physiol. 258: H1138-H1143.
4. YANKNER, B. A. 1996. New clues to Alzheimer's disease: Unraveling the roles of amyloid and tau. Nature Med. 2: 850-852.
5. YAM, S. D. et al. 1996. Rage and amyloid-β peptide neurotoxicity in Alzheimer's disease. Nature 382: 685-691.