Fc(epsilon)RI-(beta) polymorphisms unlikely to contribute substantially to genetic risk of allergic disease

BMJ

Volumn 312, Issue 7026, 1996, Pages 311-

  Hall, I P ^a   Wheatley, A ^a   Dewar, J ^a   Wilkinson, J ^b   Morrison, J ^c  

^a QUEEN S MEDICAL CENTRE   (United Kingdom)

^b SOUTHAMPTON GENERAL HOSPITAL   (United Kingdom)

^c ST JAMES S UNIVERSITY HOSPITAL   (United Kingdom)

Author keywords

[No Author keywords available]

Indexed keywords

IMMUNOGLOBULIN E RECEPTOR;

ALLERGIC DISEASE; AMINO ACID SUBSTITUTION; ASTHMA; ATOPY; DNA POLYMORPHISM; GENETIC RISK; HUMAN; LETTER; POLYMERASE CHAIN REACTION; PRIORITY JOURNAL; GENETIC POLYMORPHISM; GENETICS; HYPERSENSITIVITY; NOTE;

ASTHMA; HUMANS; HYPERSENSITIVITY; POLYMORPHISM, GENETIC; RECEPTORS, IGE;

EID: 0030567083     PISSN: 09598138     EISSN: 14685833     Source Type: Journal    
DOI: 10.1136/bmj.312.7026.311     Document Type: Letter

References (3)

1. Hill MR, James AL, Faux JA, Ryan G, Hopkin JM, Le Souef P, et al Fc(epsilon)RI-(beta) polymorphism and risk of atopy in a general population sample. BMJ 1995; 311: 776–9. (23 September.).
2. Shirakawa TS, Li A, Dubowitz M, Dekker JW, Shaw AE, Faux JA, et al Association between atopy and variants of the (beta) sub unit of the high affinity immunoglobulin E receptor. Nat Genet 1994; 7: 125–129.
3. Hall IP, Wheatley A, Wilding P, Liggett SB. Association of Glu 27 (beta)(sub 2)-adrenoceptor polymorphism with lower airway reactivity in asthmatic subjects. Lancet 1995; 345: 1213–4.