N-acetyl-β-D-glucosaminidase excretion in calcium oxalate stone patients and its relation to the risk of stone formation

Scandinavian Journal of Urology and Nephrology

Volumn 30, Issue 6, 1996, Pages 439-443

  Winter, P ^a   Ganter, K ^a   Heimbach, D ^a   Hesse, A ^a  

^a UNIVERSITY OF BONN   (Germany)

Author keywords

Calcium oxalate stone patients; Lithogenous substances; NAG excretion; Stone formation risk

Indexed keywords

N ACETYL BETA GLUCOSAMINIDASE;

ADULT; AGED; ARTICLE; CALCIUM OXALATE STONE; CONTROLLED STUDY; FEMALE; HUMAN; MAJOR CLINICAL STUDY; MALE; PRIORITY JOURNAL; RISK ASSESSMENT; STONE FORMATION; URINARY EXCRETION; UROLITHIASIS;

EID: 0030474149     PISSN: 00365599     EISSN: None     Source Type: Journal    
DOI: 10.3109/00365599609182320     Document Type: Article

References (17)

1. Assimos DG, Boyce WH, Furr EG, et al. Selective elevation of urinary enzyme levels after extracorporeal shock wave lithotripsy. J Urol 1990; 142: 687-690.
2. Backman U. Tubular defects in patients forming calcium containing stones. Eur Urol 1978; 4: 50-56.
3. Baggio B, Gambaro G, Favaro S, Borsatti A. Prevalance of hyperoxaluria in idiopathic calcium oxalate kidney stone disease. Nephron 1983; 35: 11-14.
4. Cifuentes Delatte L, Minon-Cifuentes JLR, Medina JA. Papillary stones: calcified renal tubules in Randall's plaques. J Urol 1985; 133: 490-494.
5. Erkizan Ö, Ayder AR, Minareci S, Lekili M, Dincel C. NAG, GGT, creatinine, urea and creatinine clearance before and after ESWL. Int Urol Nephrol 1994; 26: 259-262.
6. Finlayson B. Calcium stones: some physical and clinical aspects, in: Calcium metabolism in renal failure and nephrolithiasis. Wiley & Sons, New York, 1977, pp. 337-382.
7. Hesse A, Jahnen A, Klocke K, Nolde A, Scharrel O. Nachsorge bei Harnsteinpatienten. Gustav-Fischer Verlag, 1994, pp. 37.
8. Jaeger P, Portmann L, Ginalski JM, Jaquet AF, Temler E, Burckardt P. Tubulopathy in nephrolithiasis: consequence rather than cause. Kidney Int 1986; 29: 563-571.
9. Karlin GS, Schulsinger D, Urivetsky M, Smith AD. Absence of persisting parenchymal damage after extra-corporeal shock wave lithotripsy as judged by excretion of renal tubular enzymes. J Urol 1990; 144: 13-14.
10. Khan SR, Hackett RL. Proximal tubular injury and crystallization of calcium oxalate in rate urine. Urol Res 1988; 16: 185-189.
11. Khan SR, Shevock PN, Hackett RL. Urinary enzymes and calcium oxalate urolithiasis. J Urol 1989;42: 846-849.
12. Loertscher P, Scholer A, Brunner F, Harder F, Thile G. Klinische Relevanz der N-Acetyl-Glucosaminidase-Bestimmung im Urin bei Nierentransplantatempfängern mit und ohne Cyclosporin. Schweizer Med Wschr 1982; 112: 1658-1664.
13. Makise J, Saito E, Obucchi M, Kanayama M, Harakawa K, Yoshida K. Improved kinetic rate assay of urinary N-acetyl-β-D-glucosaminidase as substrate. Clin Chem 1990; 36/2: 319-322.
14. Müller PW, MacNeil ML, Steinberg KK. Stabilization of alanine aminopeptidase, gamma glutamyltranspeptidase, and N-acetyl-β-d-glucosaminidase activity in normal urines. Arch Environ Contam Toxicol 1986; 15: 343-347.
15. Noto A, Ogawa Y, Mori S, et al. Simple, rapid spectrophotometry of urinary N-acetyl-β-D-glucosaminidase with use of a new chromogenic substance. Clin Chem 1983; 29: 1713-1716.
16. Resnick MI, Boyce WH. Low molecular weight urinary proteins and renal lithiasis. Invest Urol 1979; 16: 270-273.
17. Resnick MI, Gammon CW, Sorrell MB, Boyce WH. Calcium-binding proteins and renal lithiasis. Surgery 1980; 88: 239-243.