Coticosteroid treatment of experimental autoimmune encephalomyelitis in the Lewis rat results in loss of Vβ8.2+ and myelin basic protein-reactive cells from the spinal cord, with increased total T-cell apoptosis but reduced apoptosis of Vβ8.2+ cells

Journal of Neuroimmunology

Volumn 70, Issue 2, 1996, Pages 93-101

  McCombe, P A ^a   Nickson, I ^a   Tabi, Z ^a   Pender, M P ^a  

^a UNIVERSITY OF QUEENSLAND   (Australia)

Author keywords

Apoptosis; Corticosteroids; Experimental autoimmune encephalomyelitis; Multiple sclerosis; Recovery; T lymphocytes

Indexed keywords

CD5 ANTIGEN; CORTICOSTEROID; DEXAMETHASONE; MYELIN BASIC PROTEIN; GLUCOCORTICOID; LYMPHOCYTE ANTIGEN RECEPTOR;

ALLERGIC ENCEPHALOMYELITIS; ANIMAL EXPERIMENT; ANIMAL MODEL; ANIMAL TISSUE; APOPTOSIS; ARTICLE; CONTROLLED STUDY; FEMALE; MALE; MULTIPLE SCLEROSIS; NONHUMAN; PRIORITY JOURNAL; RAT; SPINAL CORD; SUBCUTANEOUS DRUG ADMINISTRATION; T LYMPHOCYTE; ANIMAL; CYTOLOGY; DRUG EFFECT; GENETICS; IMMUNOLOGY; LEWIS RAT; PATHOLOGY; T LYMPHOCYTE SUBPOPULATION;

ANIMALS; APOPTOSIS; DEXAMETHASONE; ENCEPHALOMYELITIS, AUTOIMMUNE, EXPERIMENTAL; FEMALE; GLUCOCORTICOIDS; MALE; MYELIN BASIC PROTEINS; RATS; RATS, INBRED LEW; RECEPTORS, ANTIGEN, T-CELL, ALPHA-BETA; SPINAL CORD; T-LYMPHOCYTE SUBSETS;

EID: 0030274316     PISSN: 01655728     EISSN: None     Source Type: Journal    
DOI: 10.1016/S0165-5728(96)00043-4     Document Type: Article

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