Acute L-triiodothyronine administration potentiates inotropic responses to β-adrenergic stimulation in the isolated perfused rat heart

Cardiovascular Research

Volumn 32, Issue 2, 1996, Pages 306-310

  Tielens, Emile T ^a,b   Forder, J R ^a   Chatham, J C ^a   Marrelli, S P ^a,c   Ladenson, P W ^a  

^a JOHNS HOPKINS UNIVERSITY SCHOOL OF MEDICINE   (United States)

^b Zuiderziekenhuis Rotterdam   (Netherlands)

^c UNIVERSITY OF TEXAS MEDICAL SCHOOL   (United States)

Author keywords

Catecholamines; Contractile function; Isoproterenol; Rat, heart; Thyroid hormone

Indexed keywords

BETA ADRENERGIC RECEPTOR; ISOPRENALINE; LIOTHYRONINE; PROPYLTHIOURACIL;

ANIMAL EXPERIMENT; ANIMAL MODEL; ANIMAL TISSUE; ARTICLE; CONCENTRATION RESPONSE; CONTROLLED STUDY; DRUG EFFECT; HEART PERFORMANCE; HYPOTHYROIDISM; INOTROPISM; MALE; NONHUMAN; PRIORITY JOURNAL; RAT;

ADRENERGIC BETA-AGONISTS; ANIMALS; DOSE-RESPONSE RELATIONSHIP, DRUG; DRUG SYNERGISM; HYPOTHYROIDISM; ISOPROTERENOL; MALE; MYOCARDIAL CONTRACTION; PERFUSION; RATS; RATS, SPRAGUE-DAWLEY; STIMULATION, CHEMICAL; TRIIODOTHYRONINE;

EID: 0030222358     PISSN: 00086363     EISSN: None     Source Type: Journal    
DOI: 10.1016/0008-6363(96)00096-X     Document Type: Article

References (15)

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