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Volumn 107, Issue 5, 1996, Pages 555-557

Mutations, molecules, and myotonia

Author keywords

[No Author keywords available]

Indexed keywords

SODIUM CHANNEL;

EID: 0030147123     PISSN: 00221295     EISSN: 15407748     Source Type: Journal    
DOI: 10.1085/jgp.107.5.555     Document Type: Article
Times cited : (2)

References (14)
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    • Carter, P.J., G. Winter, A.J. Wilkinson, and A.R. Fersht. 1984. The use of double mutants to detect structural changes in the active site of the tyrosyl-tRNA synthetase (Bacillus stearothermophilus). Cell. 38:835-840.
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  • 3
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    • Sodium channel mutations in paramyotonia congenita uncouple inactivation from activation
    • Chahine, M., A.L. George, Jr., M. Zhou, S. Ji, W. Sun, R.L. Barchi, and R. Horn. 1994. Sodium channel mutations in paramyotonia congenita uncouple inactivation from activation. Neuron. 12:281-294.
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    • Chahine, M.1    George, A.L.2    Zhou, M.3    Ji, S.4    Sun, W.5    Barchi, R.L.6    Horn, R.7
  • 4
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    • Probing S4-S5 regions of the rat brain sodium channel for the fast inactivation particle receptor site
    • (Abstr.)
    • Depp, M.R., and A.L. Goldin. 1996. Probing S4-S5 regions of the rat brain sodium channel for the fast inactivation particle receptor site. Biophys. J. 70:317a. (Abstr.)
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    • Depp, M.R.1    Goldin, A.L.2
  • 5
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    • Hyperkalemic periodic paralysis and the adult muscle sodium channel alpha-subunit gene
    • Fontaine, B., T.S. Khurana, E.P. Hoffman, G.A.P. Bruns, and J.L. Haines. 1990. Hyperkalemic periodic paralysis and the adult muscle sodium channel alpha-subunit gene. Science (Wash. DC). 250:1000-1002.
    • (1990) Science (Wash. DC) , vol.250 , pp. 1000-1002
    • Fontaine, B.1    Khurana, T.S.2    Hoffman, E.P.3    Bruns, G.A.P.4    Haines, J.L.5
  • 6
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    • + channel: Peptide and channel residues mediating molecular recognition
    • + channel: peptide and channel residues mediating molecular recognition. Neuron. 12:1377-1388.
    • (1994) Neuron , vol.12 , pp. 1377-1388
    • Goldstein, S.A.1    Pheasant, D.J.2    Miller, C.3
  • 7
    • 0029926886 scopus 로고    scopus 로고
    • Inactivation defects caused by myotonia-associated mutations in the sodium channel III-IV linker
    • Hayward, L.J., R.H. Brown, Jr., and S.C. Cannon. 1996. Inactivation defects caused by myotonia-associated mutations in the sodium channel III-IV linker. J. Gen. Physiol. 107:559-576.
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  • 8
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    • Ji, S., A.L. George, R. Horn, and R.L. Barchi. 1996. Paramyotonia congenita mutations reveal different roles for segments S3 and S4 of domain D4 in hSkMl sodium channel gating. J. Gen. Physiol. 107:183-194.
    • (1996) J. Gen. Physiol , vol.107 , pp. 183-194
    • Ji, S.1    George, A.L.2    Horn, R.3    Barchi, R.L.4
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* 이 정보는 Elsevier사의 SCOPUS DB에서 KISTI가 분석하여 추출한 것입니다.