Unique growth properties of neonatal pulmonary vascular cells: Importance of time- and site-specific responses, cell-cell interaction, and synergy

Journal of Perinatology

Volumn 16, Issue PART 2, 1996, Pages

  Dempsey, Edward C ^a  

^a UNIVERSITY OF COLORADO   (United States)

Author keywords

[No Author keywords available]

Indexed keywords

ANIMAL; ANOXIA; CELL COMMUNICATION; CELL DIVISION; CYTOLOGY; HUMAN; MECHANICAL STRESS; NEWBORN; PATHOPHYSIOLOGY; PHYSIOLOGY; PULMONARY ARTERY; REVIEW; SIGNAL TRANSDUCTION; TIME; VASCULAR SMOOTH MUSCLE;

ANIMALS; ANIMALS, NEWBORN; ANOXIA; CELL COMMUNICATION; CELL DIVISION; HUMANS; INFANT, NEWBORN; MUSCLE, SMOOTH, VASCULAR; PULMONARY ARTERY; SIGNAL TRANSDUCTION; STRESS, MECHANICAL; TIME FACTORS;

EID: 0030099842     PISSN: 07438346     EISSN: None     Source Type: Journal    
DOI: None     Document Type: Article

References (3)

1. Rabinovitch M, Konstam M, Gamble W, et al. Changes in pulmonary blood flow affect vascular response to chronic hypoxia in rats. Circ Res 1983;52:432-41.
2. Stelzner TJ, O'Brien RF, Sato K, Weil JV. Hypoxia-induced increases in pulmonary transvascular protein escape in rats. J Clin Invest 1988;82: 1840-7.
3. Dempsey EC, McMurtry IF, O'Brien RF. Protein kinase C activation allows pulmonary artery smooth muscle cells to proliferate to hypoxia. Am J Physiol 1991;260:L136-L145.