A unified model explaining the preferential repair of active over inactive genes and of the transcribed over the nontranscribed strand: A leading role for transcription factors and matrix anchorage (review)

International Journal of Oncology

Volumn 8, Issue 1, 1996, Pages 77-84

  Boulikas, Teni ^a  

^a Inst of Molecular Medical Sciences   (United States)

Author keywords

Active gene; Chromatin; Dna replication; Histone h1; HMG1; Nuclear matrix; Nucleotide excision repair; p53; Protein phosphorylation; RPA; TFIIH; Transcription factors; Transcription coupled repair

Indexed keywords

TRANSCRIPTION FACTOR;

ARTICLE; DNA REPAIR; GENE ACTIVATION; GENETIC TRANSCRIPTION; MODEL; PRIORITY JOURNAL;

EID: 0030071232     PISSN: 10196439     EISSN: None     Source Type: Journal    
DOI: None     Document Type: Article

References (2)

1. Huang J-C, Svoboda DL, Reardon JT and Sancar A: Human nuclcotidc excision nuclease removes thymine dimers from DNA by incising the 22nd phosphodiester bond 5′ and the 6th phosphodicstcr bond 3′ to the photodimer. Proc Natl Acad Sci USA 89: 3664-3668, 1992.
2. Abousekhra A, Biggerstaff M, Shivji MKK, Vilpo JA, Moncollin V, Podust VN, Protic M, Hubsher U, Egly J-M and Wood RD: Mammalian DNA nucleotide excision repair reconstituted with purified protein components. Cell 80: 859-868, 1995.