Calpain inhibitors protect against depolarization-induced neurofilament protein loss of septo-hippocampal neurons in culture

European Journal of Neuroscience

Volumn 8, Issue 2, 1996, Pages 344-352

  Kampfl, A ^a  

^a UNIVERSITY OF TEXAS MEDICAL SCHOOL   (United States)

Author keywords

Calpain; Cytoskeleton; Ischaemia; Potassium depolarization; Trauma

Indexed keywords

CALCIUM CHLORIDE; CALPASTATIN; NEUROFILAMENT PROTEIN; POTASSIUM CHLORIDE;

ANIMAL CELL; ANIMAL TISSUE; ARTICLE; CELL DEATH; CELL STRUCTURE; CONTROLLED STUDY; CYTOSKELETON; DEPOLARIZATION; EXTRACELLULAR CALCIUM; FETUS; HIPPOCAMPUS; IMMUNOBLOTTING; IMMUNOHISTOCHEMISTRY; MOLECULAR WEIGHT; NERVE CELL; NERVE CELL CULTURE; NONHUMAN; PRIORITY JOURNAL; PROTEIN DEGRADATION; RAT;

ANIMALS; CALCIUM CHLORIDE; CALPAIN; CELLS, CULTURED; CYSTEINE PROTEINASE INHIBITORS; HIPPOCAMPUS; LEUPEPTINS; MEMBRANE POTENTIALS; NERVE TISSUE PROTEINS; NEUROFILAMENT PROTEINS; NEURONS; POTASSIUM CHLORIDE; RATS; SEPTUM PELLUCIDUM; SPECTRIN;

EID: 0030070664     PISSN: 0953816X     EISSN: None     Source Type: Journal    
DOI: 10.1111/j.1460-9568.1996.tb01218.x     Document Type: Article

References (2)

1. Arai, A., Kessler, M., Lee, K. and Lynch, G. (I990) Calpain inhibitors improve the recovery of synaptic transmission from hypoxia in hippocampal slices. Brain Res., 532, 63-68.
2. Arai, A., Vanderklish, P., Kessler, M., Lee, K. and Lynch, G. (1991) A brief period of hypoxia causes proteolysis of cytoskeletal proteins in hippocampal slices. Brain Res., 555, 276-280.