CDKN2/p16 or RB alterations occur in the majority of glioblastomas and are inversely correlated

Cancer Research

Volumn 56, Issue 1, 1996, Pages 150-153

  Ueki, Keisuke ^a   Ono, Yasuhiro ^a   Henson, John W ^a   Efird, Jimmy T ^b   Von Deimling, Andreas ^c   Louis, David N ^a,b  

^a Neurosurgical and Neurology Services   (United States)

^b MASSACHUSETTS GENERAL HOSPITAL   (United States)

^c UNIVERSITY HOSPITAL BONN   (Germany)

Author keywords

[No Author keywords available]

Indexed keywords

CYCLIN DEPENDENT KINASE; CYCLINE; RETINOBLASTOMA PROTEIN;

ARTICLE; ASTROCYTOMA; CARCINOGENESIS; CELL CYCLE; CHROMOSOME 13Q; CHROMOSOME 9P; CHROMOSOME LOSS; CHROMOSOME POLYMORPHISM; CHROMOSOME SATELLITE; CONTROLLED STUDY; DNA SEQUENCE; FRAMESHIFT MUTATION; GLIOBLASTOMA; HUMAN; HUMAN TISSUE; PRIORITY JOURNAL; SINGLE STRAND CONFORMATION POLYMORPHISM; TUMOR SUPPRESSOR GENE;

BASE SEQUENCE; CARRIER PROTEINS; CELL CYCLE; CYCLIN-DEPENDENT KINASE INHIBITOR P16; GENES, TUMOR SUPPRESSOR; GLIOBLASTOMA; HUMANS; MOLECULAR SEQUENCE DATA; POLYMORPHISM, GENETIC; RETINOBLASTOMA PROTEIN; SEQUENCE ANALYSIS; TUMOR CELLS, CULTURED;

EID: 0030070484     PISSN: 00085472     EISSN: None     Source Type: Journal    
DOI: None     Document Type: Article

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