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Serum amyloid A: An acute phase apolipoprotein and precursor of AA amyloid
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Marhaug G, Dowton SB: Serum amyloid A: an acute phase apolipoprotein and precursor of AA amyloid. Baillieres Clin Rheumatol 1994, 8:553-573. Current knowledge about SAA is thoroughly presented and discussed.
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Pathogenesis of amyloidosis
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Kisilevsky R, Young ID: Pathogenesis of amyloidosis. Baillieres Clin Rheumatol 1994, 8:613-626. The pathogenesis of reactive amyloidosis is presented in a comprehensive and stimulating review.
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Badolato R, Wang JM, Murphy WJ, Lloyd AR, Michiel DF, Baussermann LL, Kelvin DJ, Oppenheim JJ: Serum amyloid A is a chemoattractant: induction of migration, adhesion, and tissue infiltration of monocytes and polymorphonuclear leukocytes. J Exp Med 1994, 180:203-209. New data describing SAA as a chemoattractant for polymorphonuclear leukocytes and monocytes are presented.
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Animal models for reactive amyloidosis
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Gruys E, Snel FW: Animal models for reactive amyloidosis. Baillieres Clin Rheumatol 1994, 8:599-611. An informative review of experimental amyloidosis.
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Gruys, E.1
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Yakar S, Kaplan B, Livneh A, Martin B, Miura K, Ali-Khan Z, Shtrasburg S, Pras M: Direct evidence for SAA deposition in tissues during murine amyloidogenesis. Scand J Immunol 1994, 40:653-658. Data providing new direct evidence for SAA deposition in tissues during murine amyloidogenesis are presented.
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16
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Cathepsin B generates the most common form of amyloid A (76 residues) as a degradation product from serum amyloid A
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Yamada T, Liepnieks JJ, Hluve-Beckmann B, Benson MD: Cathepsin B generates the most common form of amyloid A (76 residues) as a degradation product from serum amyloid A. Scand J Immunol 1995, 41:94-97. Data are presented that indicate that cathepsin B may play an important role in amyloid fibrillogenesis by converting SAA to AA.
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Yamada, T.1
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Foyn Bruun C, Rygg M, Nordstoga K, Sletten K, Marhaug G: Serum amyloid A protein in mink during endotoxin induced inflammation and amyloidogenesis. Scand J Immunol 1994, 40:337-344. In endotoxin-induced amyloidosis in mink, only SAA2 protein was recovered from hepatic amyloid fibrils.
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Native human serum amyloid P component is a single pentamer
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Sörensen U, Andersen O, Holm Nielsen E, Svehag S-E: Native human serum amyloid P component is a single pentamer. Scand J Immunol 1995, 41:263-267. The data presented indicate that native SAP component circulates as a single pentamer, not as a decamer as previously believed.
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Sörensen, U.1
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Human serum amyloid P component binds to a specific peptide in the presence of calcium
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Hamazaki H: Human serum amyloid P component binds to a specific peptide in the presence of calcium. Biochem Biophys Res Commun 1994, 1172-1178. It is shown that human SAP component binds to a carboxylterminal peptide of residues 160-204 of SAP itself in the presence of calcium.
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Hamazaki, H.1
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Binding of pentraxins to different nuclear structures: C-reactive protein binds to small nuclear ribonucleoprotein particles, serum amyloid P component binds to chromatin and nucleoli
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Pepys MB, Booth SE, Tennent GA, Butler PJG, Williams DG: Binding of pentraxins to different nuclear structures: C-reactive protein binds to small nuclear ribonucleoprotein particles, serum amyloid P component binds to chromatin and nucleoli. Clin Exp Immunol 1994, 97:152-157. Using whole acute phase serum as the protein source and confocal immunofluorescence microscopy, it was shown that C-reactive protein binds to small nuclear ribonucleoproteins, whereas the other pentraxin SAP binds to chromatin and to the nucleolus.
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Clin Exp Immunol
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Pepys, M.B.1
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22
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0027969285
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The pentraxins, C-reactive protein and serum amyloid P component, are cleared and catabolized by hepatocytes in vivo
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Hutchinson WL, Noble GE, Hawkins PN, Pepys MB: The pentraxins, C-reactive protein and serum amyloid P component, are cleared and catabolized by hepatocytes in vivo. J Clin Invest 1994, 94:1390-1396. It is documented that hepatocytes represent the single major site of pentraxin clearance and catabolism in vivo. An important consequence is that SAP bound to tissue amyloid is not degraded locally.
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Hutchinson, W.L.1
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Diagnosis and monitoring of amyloidosis
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Hawkins PN: Diagnosis and monitoring of amyloidosis. Baillieres Clin Rheumatol 1994, 8:635-639. A very comprehensive review of how to diagnose and monitor amyloidosis.
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Baillieres Clin Rheumatol
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Hawkins, P.N.1
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Proteoglycans, glycosaminoglycans and amyloid deposition
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Magnus JH, Stenstad T, Husby G: Proteoglycans, glycosaminoglycans and amyloid deposition. Baillieres Clin Rheumatol 1994, 8:575-597. The role of proteoglycans and glycosaminoglycans in amyloidogenesis is expertly reviewed.
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Baillieres Clin Rheumatol
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Magnus, J.H.1
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The epidemiology of AL and AA amyloidosis
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Simms RW, Prout MN, Cohen AS: The epidemiology of AL and AA amyloidosis. Baillieres Clin Rheumatol 1994, 8:627-634. Existing data on the epidemiology of AL and AA amyloidosis are presented and analyzed.
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Baillieres Clin Rheumatol
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Simms, R.W.1
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Clinical and therapeutic aspects of AA amyloidosis
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Hazenberg BPC, van Rijswijk MH: Clinical and therapeutic aspects of AA amyloidosis. Baillieres Clin Rheumatol 1994, 8:661-690. An excellent review of the clinical and therapeutic aspects of AA amyloidosis.
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Baillieres Clin Rheumatol
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Hazenberg, B.P.C.1
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0028708966
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Amyloidosis in rheumatoid arthritis: Clinical study of 124 histologically proven cases
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Okuda Y, Takasugi K, Oyama T, Onuma M, Oyama H: Amyloidosis in rheumatoid arthritis: clinical study of 124 histologically proven cases. Ryumachi 1994, 34:939-946. Findings in 124 histologically proven cases of amyloidosis complicating RA are reported.
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Ryumachi
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Okuda, Y.1
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Amyloidosis in children
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Woo P: Amyloidosis in children. Baillieres Clin Rheumatol 1994, 8:691-697. Amyloidosis in children is expertly reviewed.
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Woo, P.1
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Livneh A, Zemer D, Langevitz P, Laor A, Sohar E, Pras M: Colchicine treatment of AA amyloidosis of familial Mediterranean fever. Arthritis Rheum 1994, 37:1804-1811. A retrospective analysis of data abstracted from the charts of 68 familial Mediterranean fever patients with amyloidosis who presented during the period 1974 to 1992 and who were treated with colchicine and followed up for at least 5 years. It was concluded that the therapeutic dosage of colchicine for amyloidosis in familial Mediterranean fever is more than 1.5 mg/d. This dosage was effective only in patients with initial serum creatinine levels of less than 1.5 mg/dL.
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Arthritis Rheum
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Livneh, A.1
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Helin HJ, Korpela MM, Mustonen JT, Pasternack AI: Renal biopsy findings and clinicopathologic correlations in rheumatoid arthritis. Arthritis Rheum 1995, 38:242-247. A retrospective study of renal biopsy specimens from 110 patients with RA in whom the clinical renal disease was probably due to RA itself and/or to antirheumatic therapy. It was concluded that the renal morphologic lesion in RA patients with isolated proteinuria and those with hematuria cannot be accurately predicted on the basis of clinical symptoms. Renal biopsy is useful in diagnosis and in assessing therapy and prognosis.
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Helin, H.J.1
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A case of rheumatoid arthritis with renal tubular amyloidosis
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Hiki Y, Horii A, Kokubo T, Ishikawa S, Sugiyama T, Shigematsu H, Kobayashi Y: A case of rheumatoid arthritis with renal tubular amyloidosis. Nephron 1994, 68:394-395.
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Nephron
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Hiki, Y.1
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A case of Takayasu's arteritis, nephrotic syndrome, and systemic amyloidosis
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Espinosa M, Rodriguez M, Martin-Malo A, Perez R, Moreno E, Gomez JM, Blanco-Molina A. Aljama P: A case of Takayasu's arteritis, nephrotic syndrome, and systemic amyloidosis. Nephrol Dial Transplant 1994, 9:1486-1488.
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Marenco JL, Sanchez-Burson J, Ruiz Campos J, Jimenez MD, Garcia-Bragado F: Pulmonary amyloidosis and unusual lung involvement in SLE. Clin Rheumatol 1994, 13:525-527.
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Marenco, J.L.1
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The arthropathy of the Muckle-Wells syndrome
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Watts RA, Nicholls A, Scott DG: The arthropathy of the Muckle-Wells syndrome. Br J Rheumatol 1994, 33:1184-1187. Arthropathy in Muckle-Wells syndrome is described in four patients.
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Br J Rheumatol
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Watts, R.A.1
Nicholls, A.2
Scott, D.G.3
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0028944296
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Castleman disease and renal amyloidosis
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Kazes I, Deray G Jacobs C: Castleman disease and renal amyloidosis. Ann Intern Med 1995, 122:395-396. A case of Castleman disease and AA amyloidosis is reported. Resection of the lymphomatic mass caused disappearance of the nephrotic syndrome and renal amyloidosis.
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Ann Intern Med
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Kazes, I.1
Deray, G.2
Jacobs, C.3
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0028234292
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Combined treatment with cyclophosphamide and prednisolone can induce remission of nephrotic syndrome in a patient with renal amyloidosis, associated with rheumatoid arthritis
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Maezawa A, Hiromura K, Mitsuhashi H, Tsukada Y, Kanai H, Yano S, Naruse T: Combined treatment with cyclophosphamide and prednisolone can induce remission of nephrotic syndrome in a patient with renal amyloidosis, associated with rheumatoid arthritis. Clin Nephrol 1994, 42:30-32.
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Clin Nephrol
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Maezawa, A.1
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Tsukada, Y.4
Kanai, H.5
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Naruse, T.7
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37
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Remission of nephrotic syndrome in a patient with renal amyloidosis associated with Takayasu's arteritis after treatment with dimethylsulphoxide
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Makino H, Nagake Y, Murakami K, Hirakawa S, Ota Z: Remission of nephrotic syndrome in a patient with renal amyloidosis associated with Takayasu's arteritis after treatment with dimethylsulphoxide. Ann Rheum Dis 1994, 53:842.
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Combined treatment with terbutaline and aminophylline inhibits experimental amyloidosis in mice
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Brandwein S, Sipe JD, Cohen AS: Combined treatment with terbutaline and aminophylline inhibits experimental amyloidosis in mice. Arthritis Rheum 1994, 37:1757-1760. A novel pharmacologic approach to the treatment of amyloidosis is presented. Terbutaline and aminophylline, which both elevate cyclic AMP, were shown to inhibit experimental murine AA amyloidosis.
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Arthritis Rheum
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Brandwein, S.1
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Study of live donor kidney transplantation outcome in recipients with renal amyloidosis
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Sobn M, Refaie A, Moustafa F, Shokeir A, Hassan N, Sally S, Ghoneim M: Study of live donor kidney transplantation outcome in recipients with renal amyloidosis. Nephrol Dial Transplant 1994, 9:704-708. Results of live donor kidney transplantation in 16 patients with amyloidosis are presented. It is concluded that renal transplantation is a safe procedure in amyloidosis and results are similar to those obtained in patients with glomerulonephritis.
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Nephrol Dial Transplant
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Sobn, M.1
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Primary systemic amyloidosis: Clinical and laboratory features in 474 cases
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Kyle RA, Gertz MA: Primary systemic amyloidosis: clinical and laboratory features in 474 cases. Semin Hematol 1995, 32:45-59. A comprehensive and detailed review covering the clinical aspects of AL amyloidosis.
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Semin Hematol
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Kyle, R.A.1
Gertz, M.A.2
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Preferential expression of human lambda-light-chain variable-region subgroups in multiple myeloma, AL amyloidosis, and Waldenström's macroglobulinemia
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Ozaki S, Abe M, Wolfenbarger D, Weiss DT, Solomon A: Preferential expression of human lambda-light-chain variable-region subgroups in multiple myeloma, AL amyloidosis, and Waldenström's macroglobulinemia. Clin Immunol Immunopathol 1994, 71:183-189. The subgroup distribution of monoclonal λ chains in the disease states differed markedly from normal. The predominance of the λ VI subgroup among monoclonal λ chains from patients with AL amyloidosis verify previous observations that the λ VI subgroup is particularly amyloidogenic.
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Clin Immunol Immunopathol
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Ozaki, S.1
Abe, M.2
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AL-amyloidosis in monoclonal gammopathies
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Miliani A, Bergesio F: AL-amyloidosis in monoclonal gammopathies. Hematologica 1994, 79:364-366.
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Hematologica
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Miliani, A.1
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Primary systemic amyloidosis presenting as giant cell arteritis and polymyalgia rheumatica
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Salvarani C, Gabriel SE, Gertz MA, Bjornsson J, Li CY, Hunder GG: Primary systemic amyloidosis presenting as giant cell arteritis and polymyalgia rheumatica. Arthritis Rheum 1994, 17:1621-1626. Clinical features of polymyalgia rheumatica and/or giant cell arteritis may be the presenting manifestation of AL amyloidosis, as seen in this report of four patients.
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Arthritis Rheum
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Salvarani, C.1
Gabriel, S.E.2
Gertz, M.A.3
Bjornsson, J.4
Li, C.Y.5
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Sicca syndrome due to primary amyloidosis
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Schima W, Amann G, Steiner E, Steurer M, Vormittag W, Steurer M: Sicca syndrome due to primary amyloidosis. Br J Radiol 1994, 67:1023-1025.
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Br J Radiol
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Schima, W.1
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45
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0028277511
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