Ex vivo evidence for PGE2 and LTB4 involvement in cutaneous leishmaniasis : Relation with infection status and cytokine production

Parasitology

Volumn 112, Issue 1, 1996, Pages 13-19

  Milano, S ^a  

^a UNIVERSITY OF PALERMO   (Italy)

Author keywords

IFN ; Leishmania major; LTB4; PGE2; TNF

Indexed keywords

GAMMA INTERFERON; LEUKOTRIENE B4; PROSTAGLANDIN E2; TUMOR NECROSIS FACTOR ALPHA;

ANIMAL CELL; ANIMAL EXPERIMENT; ARTICLE; CONTROLLED STUDY; CYTOKINESIS; LEISHMANIA MAJOR; MOUSE; NONHUMAN; PRIORITY JOURNAL; SKIN LEISHMANIASIS; SPLEEN CELL;

ANIMALS; CELLS, CULTURED; DINOPROSTONE; FEMALE; INTERFERON TYPE II; INTERLEUKIN-4; LEISHMANIA MAJOR; LEISHMANIASIS, CUTANEOUS; LEUKOTRIENE B4; MICE; MICE, INBRED BALB C; SPLEEN; TH1 CELLS; TH2 CELLS; TUMOR NECROSIS FACTOR-ALPHA; WHOLE-BODY IRRADIATION;

EID: 0030027099     PISSN: 00311820     EISSN: None     Source Type: Journal    
DOI: 10.1017/s0031182000065033     Document Type: Article

References (2)

1. AKAHOSHI, T., OPPENHEIM, J. J. & MATSUSHIMA, K. (1988). Interleukin-l stimulates its own receptor expression on human fibroblast through the endogenous production of prostaglandin(s). Journal of Clinical Investigation 82, 1219-24.
2. 2 lymphokines. International Journal of Immunopharmacology 17, 457-63.