Mouse lung tumors exhibit specific Ki-ras mutations following transplacental exposure to 3-methylcholanthrene

Carcinogenesis

Volumn 17, Issue 7, 1996, Pages 1519-1526

  Wessner, Lisa L ^a   Fan, Manxia ^a   Schaeffer, Dorcas O ^a   McEntee, Michael F ^a   Miller, Mark Steven ^a,b,c  

^a UNIVERSITY OF TENNESSEE   (United States)

^b UNIVERSITY OF TENNESSEE MEDICAL CENTER   (United States)

^c WAKE FOREST SCHOOL OF MEDICINE   (United States)

Author keywords

[No Author keywords available]

Indexed keywords

3 METHYLCHOLANTHRENE; OLIVE OIL; POLYCYCLIC AROMATIC HYDROCARBON;

ANIMAL EXPERIMENT; ANIMAL MODEL; ANIMAL TISSUE; ARTICLE; CODON; CONTROLLED STUDY; EXON; FEMALE; LUNG CARCINOGENESIS; MALE; MOUSE; NONHUMAN; ONCOGENE K RAS; PHARMACOGENETICS; POINT MUTATION; POLYMERASE CHAIN REACTION; PRIORITY JOURNAL; TRANSPLACENTAL CARCINOGENESIS;

ANIMALS; BASE SEQUENCE; BODY WEIGHT; CARCINOGENS; CROSSES, GENETIC; CYTOCHROME P-450 CYP1A1; CYTOCHROME P-450 ENZYME SYSTEM; DNA PRIMERS; FEMALE; GENES, RAS; LUNG NEOPLASMS; MALE; METHYLCHOLANTHRENE; MICE; MICE, INBRED DBA; MICE, INBRED STRAINS; MOLECULAR SEQUENCE DATA; OXIDOREDUCTASES; PHENOTYPE; PREGNANCY; PRENATAL EXPOSURE DELAYED EFFECTS; RECEPTORS, ARYL HYDROCARBON; SEX CHARACTERISTICS;

EID: 0029987470     PISSN: 01433334     EISSN: None     Source Type: Journal    
DOI: 10.1093/carcin/17.7.1519     Document Type: Article

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