Release of soluble intercellular adhesion molecule 1 into bile and serum in murine endotoxin shock

Hepatology

Volumn 23, Issue 3, 1996, Pages 530-536

  Jaeschke, Hartmut ^a   Essani, Naeem A ^a   Fisher, Michael A ^a   Vonderfecht, Steven L ^a   Farhood, Anwar ^b   Wayne Smith, C ^c  

^a PFIZER INC   (United States)

^b UNIVERSITY OF TEXAS   (United States)

^c BAYLOR COLLEGE OF MEDICINE   (United States)

Author keywords

[No Author keywords available]

Indexed keywords

GALACTOSAMINE; INTERCELLULAR ADHESION MOLECULE 1; SALMONELLA ENDOTOXIN;

ANIMAL EXPERIMENT; ANIMAL MODEL; ARTICLE; BILE FLOW; BILE SECRETION; CELL ADHESION; CONTROLLED STUDY; ENDOTHELIUM CELL; ENDOTOXEMIA; GENE INDUCTION; LIVER INJURY; LIVER ISCHEMIA; MOUSE; NONHUMAN; PHAGOCYTOSIS; PRIORITY JOURNAL; RECEPTOR UPREGULATION; SEPTIC SHOCK;

EID: 0029980411     PISSN: 02709139     EISSN: None     Source Type: Journal    
DOI: 10.1002/hep.510230318     Document Type: Article

References (7)

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4. Vedder NB, Winn RK, Rice CL, Chi EY, Arfors KE, Harlan JM. A monoclonal antibody to the adherence-promoting leukocyte glycoprotein, CD 18, reduces organ injury and improves survival from hemorrhagic shock and resuscitation in rabbits. J Clin Invest 1988;81:939-944.
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7. Witthaut R, Farhood A, Smith CW, Jaeschke H. Complement and tumor necrosis factor-α contribute to Mac-1 (CD11b/CD18) up-regulation and systemic neutrophil activation during endotoxemia in vivo. J Leukoc Biol 1994;55:105-111.