Abrogation of p53 function affects gadd gene responses to DNA base- damaging agents and starvation

DNA and Cell Biology

Volumn 15, Issue 10, 1996, Pages 805-815

  Zhan, Qimin ^a  

^a Laboratory of Molecular Pharmacology   (United States)

Author keywords

[No Author keywords available]

Indexed keywords

PROTEIN P53;

ALLELE; ARTICLE; DNA DAMAGE; DNA STRAND BREAKAGE; EFFECTOR CELL; GENE FUNCTION; GENE INDUCTION; GENETIC REGULATION; HUMAN; HUMAN CELL; INTRON; IONIZING RADIATION; PRIORITY JOURNAL; PROTEIN STABILITY; STARVATION; TUMOR SUPPRESSOR GENE;

MAMMALIA; MINK CELL FOCUS-FORMING VIRUS;

EID: 0029960486     PISSN: 10445498     EISSN: None     Source Type: Journal    
DOI: 10.1089/dna.1996.15.805     Document Type: Article

References (3)

1. BAKER, S.J., MARKOWITZ, S., FEARON, E.R., WILLSON, J.K., and VOGELSTEIN, B. (1990). Suppression of human colorectal carcinoma cell growth by wild-type p53. Science 249, 912-915.
2. BARAK, Y., and OREN, M. (1992). Enhanced binding of a 95 kDa protein to p53 in cells undergoing p53-mediated growth arrest. EMBO J. 11, 2115-2121.
3. CAELLES, C., HEIMBERG, A., and KARIN, M. (1994). p53-dependent apoptosis in the absence of transcriptional activation of p53-target genes. Nature 370, 220-223.