The molecular basis of inherited hypokalemic alkalosis: Bartter's and Gitelman's syndromes

American Journal of Physiology

Volumn 271, Issue 5 PART 2, 1996, Pages

  Simon, David B ^a,b,c   Lifton, Richard P ^a  

^a HOWARD HUGHES MEDICAL INSTITUTE   (United States)

^b Division of Nephrology   (United States)

^c YALE UNIVERSITY SCHOOL OF MEDICINE   (United States)

Author keywords

Electrolyte homeostasis; Hypokalemia; Hypomagnesemia; Renal physiology

Indexed keywords

BUMETANIDE; CARRIER PROTEIN; COMPLEMENTARY DNA; THIAZIDE DIURETIC AGENT;

AUTOSOMAL RECESSIVE DISORDER; BARTTER SYNDROME; CHLORIDE TRANSPORT; CLINICAL FEATURE; DNA SEQUENCE; ELECTROLYTE BALANCE; GENE LOCUS; GENE MUTATION; HUMAN; HYPOKALEMIC ALKALOSIS; KIDNEY FUNCTION; KIDNEY TUBULE DISORDER; MOLECULAR CLONING; PATHOPHYSIOLOGY; PHENOTYPE; POTASSIUM TRANSPORT; PRIORITY JOURNAL; REVIEW; SODIUM TRANSPORT;

ALKALOSIS; ANIMALS; BARTTER SYNDROME; CARRIER PROTEINS; GENES; HUMANS; HYPOKALEMIA; LINKAGE (GENETICS); MUTATION; SODIUM CHLORIDE SYMPORTERS; SODIUM-POTASSIUM-CHLORIDE SYMPORTERS; SYMPORTERS; SYNDROME;

EID: 0029951224     PISSN: 00029513     EISSN: None     Source Type: Journal    
DOI: 10.1152/ajprenal.1996.271.5.f961     Document Type: Article

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