Lactic acidosis during sepsis is related to increased pyruvate production, not deficits in tissue oxygen availability

Annals of Surgery

Volumn 224, Issue 1, 1996, Pages 97-102

  Gore, Dennis C ^a   Jahoor, Farook ^b   Hibbert, Jacqueline M ^a   DeMaria, Eric J ^a  

^a VIRGINIA COMMONWEALTH UNIVERSITY SCHOOL OF MEDICINE   (United States)

^b BAYLOR COLLEGE OF MEDICINE   (United States)

Author keywords

[No Author keywords available]

Indexed keywords

DICHLOROACETIC ACID; LACTIC ACID; PYRUVIC ACID;

ADULT; ARTICLE; CARBOHYDRATE METABOLISM; CLINICAL ARTICLE; FEMALE; HUMAN; LACTATE BLOOD LEVEL; LACTIC ACIDOSIS; OXYGEN CONSUMPTION; PRIORITY JOURNAL; SEPSIS; TISSUE OXYGENATION;

ACIDOSIS, LACTIC; ADULT; BLOOD GLUCOSE; BREATH TESTS; CALORIMETRY, INDIRECT; DICHLOROACETATE; FEMALE; HUMANS; LACTATES; MIDDLE AGED; OXYGEN; OXYGEN CONSUMPTION; PYRUVATES; SEPSIS; TIME FACTORS;

EID: 0029947642     PISSN: 00034932     EISSN: None     Source Type: Journal    
DOI: 10.1097/00000658-199607000-00015     Document Type: Article

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