Brief report: Genetic, prenatal, and immunologic factors

Journal of Autism and Developmental Disorders

Volumn 26, Issue 2, 1996, Pages 195-198

  Smalley, Susan L ^b   Collins, Francis ^a  

^a National Human Genome Institute   (United States)

^b UNIVERSITY OF CALIFORNIA   (United States)

Author keywords

[No Author keywords available]

Indexed keywords

AUTISM; CONFERENCE PAPER; GENETIC DISORDER; GENETIC LINKAGE; GENOTYPE; HUMAN; PATHOGENESIS; PHENOTYPE; PRIORITY JOURNAL;

AUTISTIC DISORDER; CHILD; CHILD, PRESCHOOL; FEMALE; HUMANS; INFANT; INFANT, NEWBORN; MALE; PREGNANCY; PRENATAL EXPOSURE DELAYED EFFECTS; RISK FACTORS; TWIN STUDIES;

EID: 0029944752     PISSN: 01623257     EISSN: None     Source Type: Journal    
DOI: 10.1007/BF02172011     Document Type: Conference Paper

References (16)

1. Bailey, A., Le Couleur, A., Gottesman, L., Bolton, P., Simonoff, E., Yuzda, E., & Rutter, M. (1995). Autism as a strongly genetic disorder: Evidence from a British twin study. Psychological Medicine, 25, 63-77.
2. Bolton, P., MacDonald, H., Pickles, A., Rios, P., Goode, S., Crowson, M., Bailey, A., & Rutter, M. (1994). A case-control family history study of autism. Journal of Child Psychology and Psychiatry, 35, 877-900.
3. Collins, F. S. (1995). Positional cloning moves from perditional to traditional. Nature Genetics, 9, 347-350.
4. Gyapay, G., Morissette, J., Vignal, A., Dib, C., Fizames, C., Millasseau, P., Marc, S., Bernardi, G., Lathrop, G., & Weissenbach, J. (1994). The 1993-1994 Genethon human genetic linkage map. Nature Genetics, 7, 246-339.
5. Lander, E., & Kruglyak, L. (1995). Genetic dissection of complex traits: Guidelines for interpreting and reporting linkage results. Nature Genetics, 11, 241-247.
6. Lander, E. S., & Schork, N.J. (1994). Genetic dissection of complex traits. Science, 265, 2037-2048.
7. Murray, J. C., Buetow, K. H., Weber, J. L., Ludwigsen, S., Scherpbier-Heddema, T., et al. (1994). A comprehensive human linkage map with Centimorgan Density. Science, 265, 2049-2054.
8. Nelson, K. B. (1991). Prenatal and perinatal factors in the etiology of autism. Pediatrics, 87, 761-766.
9. Pickles, A., Bolton, P., Macdonald, H., Bailey, A., Le Couleur, A., Sim, C.-H., & Rutter, M. (1995). Latent-class analysis of recurrence risks for complex phenotypes with selection and measurement error. A twin and family history study of autism. American Journal of Human Genetics, 57, 717-726.
10. Piven, J., Chase, G. A., Landa, R., Wzorek, M., Gayle, J., Cloud, D., & Folstein, S. (1991). Psychiatric disorders in the parents of autistic individuals. Journal of the American Academy of Child and Adolescent Psychiatry, 30, 472-478.
11. Pritchard, L. E., Kawaguchi, Y., Reed, P. W., Copeman, J. B., Davies, J. L., Barnett, A. H., Bain, S. C., & Todd, J. A. (1995). Analysis of the CD3 gene region and type I diabetes: Application of fluorescence-based technology to linkage disequilibrium mapping. Human Molecular Genetics, 4, 197-202.
12. Risch, N. (1990). Linkage strategies for genetically complex traits: II. The power of affected relative pairs. American Journal of Human Genetics, 46, 229-241.
13. Rutter, M., Bailey, A., Bolton., P., & Le Couteur, A. (1994). Autism and known medical conditions: Myth and substance. Journal of Child Psychology and Psychiatry, 35, 311-322.
14. Smalley, S. L., Asarnow, R. F., & Spence, M. A. (1988). Autism and genetics. Archives of General Psychiatry, 45, 953-961.
15. Todd, J. A. (1995). Genetic analysis of type I diabetes using whole genome approaches. Proceedings of the National Academy of Sciences, U.S., 92, 8560-8565.
16. Warren, R. P., Yonk, L. J., Burger, R. A., Cole, P., Odell, J. D., et al. (1990). Deficiency of suppressor-inducer (CD4+CD45RA+) T cells in autism. Immunological Investigations, 19, 245-251.