GAP-43 mRNA in mouse motoneurons undergoing axonal sprouting in response to muscle paralysis or partial denervation

European Journal of Neuroscience

Volumn 8, Issue 6, 1996, Pages 1240-1248

  Bisby, M A ^a  

^a QUEEN S UNIVERSITY   (Canada)

Author keywords

Botulinum toxin; In situ hybridization; Marcaine; Plasticity

Indexed keywords

BOTULINUM TOXIN; BUPIVACAINE; COMPLEMENTARY DNA; MESSENGER RNA; NEUROMODULIN;

ANIMAL EXPERIMENT; ANIMAL TISSUE; ARTICLE; DENERVATION; FEMALE; IN SITU HYBRIDIZATION; MALE; MOTONEURON; MOUSE; NERVE FIBER; NERVE FIBER TRANSECTION; NONHUMAN; OLIGONUCLEOTIDE PROBE; PARALYSIS; PRIORITY JOURNAL;

ANIMALS; AXONS; BASE SEQUENCE; BUPIVACAINE; GAP-43 PROTEIN; GROWTH SUBSTANCES; MEMBRANE GLYCOPROTEINS; MICE; MOLECULAR SEQUENCE DATA; MOTOR NEURONS; MUSCLE DENERVATION; NERVE TISSUE PROTEINS; PARALYSIS;

EID: 0029930709     PISSN: 0953816X     EISSN: None     Source Type: Journal    
DOI: 10.1111/j.1460-9568.1996.tb01292.x     Document Type: Article

References (1)

1. Aigner, L. and Caroni, P. (1993) Depletion of 43-kD growth-associated protein in primary sensory neurons leads to diminished formation and spreading of growth cones. J. Cell Biol, 123, 417-129.