Cloning and characterization of the bovine Fas

DNA and Cell Biology

Volumn 15, Issue 3, 1996, Pages 227-234

  Yoo, Jakyoung ^a,b   Stone, Roger T ^a   Beattie, Craig W ^a  

^a UNIVERSITY OF CALIFORNIA   (United States)

^b U S MEAT ANIMAL RESEARCH CENTER   (United States)

Author keywords

[No Author keywords available]

Indexed keywords

TUMOR NECROSIS FACTOR;

AMINO ACID SEQUENCE; ANIMAL CELL; APOPTOSIS; ARTICLE; CATTLE; CELL CULTURE; CELL DEATH; CELL MEMBRANE; GENETIC TRANSFECTION; IMMUNOBLOTTING; NONHUMAN; NUCLEOTIDE SEQUENCE; PRIORITY JOURNAL; PROTEIN ANALYSIS;

ANIMALIA; BOS TAURUS; BOVINAE;

EID: 0029926569     PISSN: 10445498     EISSN: None     Source Type: Journal    
DOI: 10.1089/dna.1996.15.227     Document Type: Article

References (3)

1. ADACHI, M., WATANABE-FUKUNAGA, R., and NAGATA, S. (1993). Aberrant transcription caused by the insertion of an early transposable element in an intron of the Fas antigen gene of Ipr mice. Proc. Natl. Acad. Sci. USA 90, 1756-1760.
2. BOLDIN, M.P., VARFOLOMEEV, E.E., PANCER, Z., METT, I.L., CAMONIS, J.H., and WALLACH, D. (1995a). A novel protein that interacts with the death domain of Fas/APO-1 contains a sequence motif related to the death domain. J. Biol. Chem. 270, 7795-7798.
3. BRUNNER, T., MOGIL, R J., LAFACE, D., YOO, N.J., MAHBOUBI, A., ECHEVERRI, F., MARTIN, S.J., FORCE, W.R., LYNCH, D.H., WARE, CF., and GREEN, D.R. (1995). Cell-autonomous Fas (CD95)/Fas-ligand interaction mediates activation-induced apoptosis in T-cell hybridomas. Nature 373, 441-444.