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Molecular rearrangements on chromosome 11q23 predominate in infant acute lymphoblastic leukemia and are associated with specific biologic variables and poor outcome
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Clonal, nonconstitutional rearrangements of the MLL gene in infant twins with acute Iymphoblastic leukemia: In utero chromosome rearrangement of 11q23
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p53 gene inactivation in acute lymphoblastic leukemia of B cell lineage associates with chromosomal breakpoints at 11q23 and 8q24
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