The immunopathologic and inflammatory differences between dermatomyositis, polymyositis and sporadic inclusion body myositis

Current Opinion in Neurology

Volumn 9, Issue 3, 1996, Pages 235-239

  Dalakas, Marinos C ^a,b   Sivakumar, K ^a  

^a NATIONAL INSTITUTE OF NEUROLOGICAL DISORDERS AND STROKE   (United States)

^b Building 10 ^*  (United States)

Author keywords

[No Author keywords available]

Indexed keywords

AUTOANTIBODY; CELL ADHESION MOLECULE; CYTOKINE; MUSCLE ANTIGEN; T LYMPHOCYTE RECEPTOR;

CELL INCLUSION; COMPLEMENT SYSTEM; DERMATOMYOSITIS; DERMIS; ENDOTHELIUM CELL; GENE EXPRESSION; HUMAN; IMMUNOPATHOGENESIS; MACROPHAGE; MYOSITIS; NATURAL KILLER CELL; POLYMYOSITIS; RECEPTOR GENE; REVIEW; T LYMPHOCYTE;

EID: 0029908563     PISSN: 13507540     EISSN: None     Source Type: Journal    
DOI: 10.1097/00019052-199606000-00015     Document Type: Review

References (34)

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14. + T cells. J Exp Med 1995, 181:1863-1868. A combination of PCR and double-fluorescence immunocytochemistry were used to analyse the TCR repertoire expressed among autoinvasive and nonautoinvasive T-cells in polymyositis patients. The results suggest that a strikingly limited TCR repertoire is expressed in polymyositic muscle; with dissociation between the TCR usage of autoinvasive and interstitial T-cells with clonal expansion of autoinvasive T-cells.
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