A point mutation in the HIV-1 Tat responsive element is associated with postintegration latency

Proceedings of the National Academy of Sciences of the United States of America

Volumn 93, Issue 13, 1996, Pages 6377-6381

  Emiliani, Stephane ^a   Van Lint, Carine ^a   Fischle, Wolfgang ^a   Paras Jr , Peter ^b   Ott, Melanie ^a   Brady, John ^c   Verdin, Eric ^a  

^a PICOWER INST FOR MEDICAL RESEARCH   (United States)

^b NATIONAL INSTITUTE OF NEUROLOGICAL DISORDERS AND STROKE   (United States)

^c NATIONAL CANCER INSTITUTE   (United States)

Author keywords

[No Author keywords available]

Indexed keywords

TRANSACTIVATOR PROTEIN;

ARTICLE; DNA SEQUENCE; GENETIC COMPLEMENTATION; HUMAN; HUMAN CELL; HUMAN IMMUNODEFICIENCY VIRUS 1; LONG TERMINAL REPEAT; PHENOTYPE; POINT MUTATION; PRIORITY JOURNAL; TRANSACTIVATION; VIRUS DNA CELL DNA INTERACTION; VIRUS LATENCY; VIRUS REPLICATION;

BASE SEQUENCE; DNA PRIMERS; GENE PRODUCTS, TAT; HIV LONG TERMINAL REPEAT; HIV-1; MOLECULAR SEQUENCE DATA; MUTAGENESIS, SITE-DIRECTED; PHENOTYPE; POINT MUTATION; PROVIRUSES; TRANSFECTION; VIRUS LATENCY; VIRUS REPLICATION;

EID: 0029901662     PISSN: 00278424     EISSN: None     Source Type: Journal    
DOI: 10.1073/pnas.93.13.6377     Document Type: Article

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