-
1
-
-
0027508926
-
Alzheimer disease amyloid β protein forms calcium channels in bilayer membranes: Blockade by tromthamine and aluminium
-
Arispe N., Rojas E., and Pollard B. (1993) Alzheimer disease amyloid β protein forms calcium channels in bilayer membranes: blockade by tromthamine and aluminium. Proc. Natl. Acad. Sci. USA 90, 567-571.
-
(1993)
Proc. Natl. Acad. Sci. USA
, vol.90
, pp. 567-571
-
-
Arispe, N.1
Rojas, E.2
Pollard, B.3
-
2
-
-
0026506810
-
+-induced depolarization selectively promote the expression of an NMDA-preferring class of ionotropic glutamate receptors in cerebellar granule neurones
-
+-induced depolarization selectively promote the expression of an NMDA-preferring class of ionotropic glutamate receptors in cerebellar granule neurones. Neurosci. Lett. 137, 109-113.
-
(1992)
Neurosci. Lett.
, vol.137
, pp. 109-113
-
-
Balazs, R.1
Resink, A.2
Hack, N.3
Van Der Valk, J.B.F.4
Kumar, K.N.5
Michaelis, E.6
-
3
-
-
0028233494
-
Hydrogen peroxide mediates amyloid β protein toxicity
-
Behl C., Davis J. B., Lesley R., and Schubert D. (1994) Hydrogen peroxide mediates amyloid β protein toxicity. Cell 77, 817-827.
-
(1994)
Cell
, vol.77
, pp. 817-827
-
-
Behl, C.1
Davis, J.B.2
Lesley, R.3
Schubert, D.4
-
4
-
-
0026745610
-
Mutation of the β-amyloid precursor protein in familial Alzheimer's disease causes increased β-protein production
-
Citron M. T., Oltersdorf T., Haass C., McConglogue L., Hung A. Y., Seubert P., Vigo-Pelfrey C., Lieberburg I., and Selkoe D. J. (1993) Mutation of the β-amyloid precursor protein in familial Alzheimer's disease causes increased β-protein production. Nature 360, 672-674.
-
(1993)
Nature
, vol.360
, pp. 672-674
-
-
Citron, M.T.1
Oltersdorf, T.2
Haass, C.3
McConglogue, L.4
Hung, A.Y.5
Seubert, P.6
Vigo-Pelfrey, C.7
Lieberburg, I.8
Selkoe, D.J.9
-
5
-
-
0025277521
-
Differential expression of excitatory amino acid receptor subtypes in cultured cerebellar neurons
-
Cox J. A., Felder C C., and Henneberry R. C. (1990) Differential expression of excitatory amino acid receptor subtypes in cultured cerebellar neurons. Neuron 4, 941-947.
-
(1990)
Neuron
, vol.4
, pp. 941-947
-
-
Cox, J.A.1
Felder, C.C.2
Henneberry, R.C.3
-
7
-
-
0027191973
-
Apoptosis mediated neuronal degeneration induced by chronic application of β amyloid fragment 25-35
-
Forloni G , Chiesa R., Smiroldo S., Verga L., Salmona M., Tagliavini F., and Angeretti N. (1993) Apoptosis mediated neuronal degeneration induced by chronic application of β amyloid fragment 25-35. Neuroreport 4, 523-526.
-
(1993)
Neuroreport
, vol.4
, pp. 523-526
-
-
Forloni, G.1
Chiesa, R.2
Smiroldo, S.3
Verga, L.4
Salmona, M.5
Tagliavini, F.6
Angeretti, N.7
-
8
-
-
0028924891
-
Apoptosis in cerebellar granule cells is blocked by high KCl, forskolin, and IGF-1 through distinct mechanisms of action: The involvement of intracellular calcium and RNA synthesis
-
Galli C., Meucci O., Scorziello A., Werge T. M., Calissano P., and Schettini G. (1995) Apoptosis in cerebellar granule cells is blocked by high KCl, forskolin, and IGF-1 through distinct mechanisms of action: the involvement of intracellular calcium and RNA synthesis. J. Neurosci. 15, 1172-1179.
-
(1995)
J. Neurosci.
, vol.15
, pp. 1172-1179
-
-
Galli, C.1
Meucci, O.2
Scorziello, A.3
Werge, T.M.4
Calissano, P.5
Schettini, G.6
-
9
-
-
0023244774
-
The role of depolarization in the survival and differentiation of cerebellar granule cells in culture
-
Gallo V., Kingsbury A., Balazs R., and Jergensen O. S. (1987) The role of depolarization in the survival and differentiation of cerebellar granule cells in culture. J. Neurosci. 7, 2203-2213.
-
(1987)
J. Neurosci.
, vol.7
, pp. 2203-2213
-
-
Gallo, V.1
Kingsbury, A.2
Balazs, R.3
Jergensen, O.S.4
-
10
-
-
0028169905
-
Secreted forms of β-amyloid precursor protein protect hippocampal neurons against amyloid β-peptide induced oxidative injury
-
Goodman Y. and Mattson M P. (1994) Secreted forms of β-amyloid precursor protein protect hippocampal neurons against amyloid β-peptide induced oxidative injury. Exp. Neurol. 128, 1-12.
-
(1994)
Exp. Neurol.
, vol.128
, pp. 1-12
-
-
Goodman, Y.1
Mattson, M.P.2
-
11
-
-
0021895138
-
A new generation of Ca2+ indicators with greatly improved fluorescence properties
-
Grynkiewicz G., Poenie M., and Tsien R. Y. (1985) A new generation of Ca2+ indicators with greatly improved fluorescence properties. J. Biol. Chem. 260, 3440-3450.
-
(1985)
J. Biol. Chem.
, vol.260
, pp. 3440-3450
-
-
Grynkiewicz, G.1
Poenie, M.2
Tsien, R.Y.3
-
12
-
-
0026646604
-
Amyloid β-peptide is produced by cultured cells during normal metabolism
-
Haass C., Schlossmacher M. G., Hung A. Y., Vigo-Pelfrey C., Mellon A., Ostaszewski B. L., Lieberburg I., Koo E H., Schenk D., Teplow D. B., and Selkoe D. J. (1992) Amyloid β-peptide is produced by cultured cells during normal metabolism. Nature 359, 322-325.
-
(1992)
Nature
, vol.359
, pp. 322-325
-
-
Haass, C.1
Schlossmacher, M.G.2
Hung, A.Y.3
Vigo-Pelfrey, C.4
Mellon, A.5
Ostaszewski, B.L.6
Lieberburg, I.7
Koo, E.H.8
Schenk, D.9
Teplow, D.B.10
Selkoe, D.J.11
-
13
-
-
0028180518
-
A model for β-amyloid aggregation and neurotoxicity based on free radical generation by the peptide: Relevance to Alzheimer disease
-
Hensley K., Carney J. M., Mattson M. P., Aksenova M., Harris M., Wu J. F., Floyd R. A., and Butterfield D. A. (1994) A model for β-amyloid aggregation and neurotoxicity based on free radical generation by the peptide: relevance to Alzheimer disease. Proc. Natl. Acad. Sci. USA 91, 3270-3274.
-
(1994)
Proc. Natl. Acad. Sci. USA
, vol.91
, pp. 3270-3274
-
-
Hensley, K.1
Carney, J.M.2
Mattson, M.P.3
Aksenova, M.4
Harris, M.5
Wu, J.F.6
Floyd, R.A.7
Butterfield, D.A.8
-
14
-
-
0025110182
-
β-Amyloid protein increases the vulnerability of cultured cortical neurons to excitotoxic damage
-
Koh J. Y., Yang L. L., and Cotman C. W. (1990) β-Amyloid protein increases the vulnerability of cultured cortical neurons to excitotoxic damage. Brain Res. 533, 315-320.
-
(1990)
Brain Res.
, vol.533
, pp. 315-320
-
-
Koh, J.Y.1
Yang, L.L.2
Cotman, C.W.3
-
15
-
-
0025879957
-
An in vivo model for the neurodegenerative effects of β amyloid and protection by substance P
-
Kowall N. W., Beal M. F., Busciglio J., Duffy L. W., and Yankner B. A. (1991) An in vivo model for the neurodegenerative effects of β amyloid and protection by substance P. Proc. Natl. Acad. Sci. USA 88, 7247-7251.
-
(1991)
Proc. Natl. Acad. Sci. USA
, vol.88
, pp. 7247-7251
-
-
Kowall, N.W.1
Beal, M.F.2
Busciglio, J.3
Duffy, L.W.4
Yankner, B.A.5
-
16
-
-
0015519878
-
The effect of potassium on neuronal differentiation in cultures of dissociated newborn rat cerebellum
-
Lasher R. S. and Zagon I. S. (1972) The effect of potassium on neuronal differentiation in cultures of dissociated newborn rat cerebellum. Brain Res. 41, 482-488.
-
(1972)
Brain Res.
, vol.41
, pp. 482-488
-
-
Lasher, R.S.1
Zagon, I.S.2
-
17
-
-
0021335259
-
Autoradiographic localization and depolarization-induced release of acidic amino acids in differentiating cerebellar granule cell cultures
-
Levi G., Aloisi F., Ciotti M. T., and Gallo V. (1984) Autoradiographic localization and depolarization-induced release of acidic amino acids in differentiating cerebellar granule cell cultures. Brain Res. 290, 77-86.
-
(1984)
Brain Res.
, vol.290
, pp. 77-86
-
-
Levi, G.1
Aloisi, F.2
Ciotti, M.T.3
Gallo, V.4
-
18
-
-
0027257708
-
Apoptosis is induced by β-amyloid in cultured central nervous system neurons
-
Loo D. T., Copani A., Pike C. J., Whittemore E. R., Walencewicz A. J., and Cotman C. W. (1993) Apoptosis is induced by β-amyloid in cultured central nervous system neurons. Proc. Natl. Acad. Sci. USA 90, 7951-7955.
-
(1993)
Proc. Natl. Acad. Sci. USA
, vol.90
, pp. 7951-7955
-
-
Loo, D.T.1
Copani, A.2
Pike, C.J.3
Whittemore, E.R.4
Walencewicz, A.J.5
Cotman, C.W.6
-
19
-
-
0024475202
-
Delayed increase of Ca2+ influx elicited by glutamate: Role in neuronal death
-
Manev H., Favaron M., Guidotti A., and Costa E. (1989) Delayed increase of Ca2+ influx elicited by glutamate: role in neuronal death. Mol. Pharmacol. 36, 106-112.
-
(1989)
Mol. Pharmacol.
, vol.36
, pp. 106-112
-
-
Manev, H.1
Favaron, M.2
Guidotti, A.3
Costa, E.4
-
20
-
-
0026570528
-
β-Amyloid peptides destabilize calcium homeostasis and render human cortical neurons vulnerable to excitotoxicity
-
Mattson M. P., Cheng B., Davis D., Bryant K., Lieberburg I., and Rydel R. (1992) β-Amyloid peptides destabilize calcium homeostasis and render human cortical neurons vulnerable to excitotoxicity. J. Neurosci. 12, 376-389.
-
(1992)
J. Neurosci.
, vol.12
, pp. 376-389
-
-
Mattson, M.P.1
Cheng, B.2
Davis, D.3
Bryant, K.4
Lieberburg, I.5
Rydel, R.6
-
21
-
-
0027297138
-
Calcium-destabilizing and neurodegenerative effects of aggregated β-amyloid peptide are attenuated by basic FGF
-
Mattson M. P., Tomaselli K. J., and Rydel R. E (1993a) Calcium-destabilizing and neurodegenerative effects of aggregated β-amyloid peptide are attenuated by basic FGF. Brain Res. 621, 35-49.
-
(1993)
Brain Res.
, vol.621
, pp. 35-49
-
-
Mattson, M.P.1
Tomaselli, K.J.2
Rydel, R.E.3
-
22
-
-
0027367765
-
β-Amyloid precursor protein metabolites and loss of neuronal Ca2+ homeostasis in Alzheimer's disease
-
Mattson M. P., Barger S. W., Cheng B., Lieberburg I., Smith-Swintosky V., and Rydel R. E. (1993b) β-Amyloid precursor protein metabolites and loss of neuronal Ca2+ homeostasis in Alzheimer's disease. Trends Neurosci. 16, 409-414.
-
(1993)
Trends Neurosci.
, vol.16
, pp. 409-414
-
-
Mattson, M.P.1
Barger, S.W.2
Cheng, B.3
Lieberburg, I.4
Smith-Swintosky, V.5
Rydel, R.E.6
-
23
-
-
0025257513
-
Physiological and pathophysiological roles of excitatory amino acids during central nervous system development
-
McDonald J. W. and Johnston M. V. (1990) Physiological and pathophysiological roles of excitatory amino acids during central nervous system development. Brain Res. Rev. 15, 41-70.
-
(1990)
Brain Res. Rev.
, vol.15
, pp. 41-70
-
-
McDonald, J.W.1
Johnston, M.V.2
-
24
-
-
0025992417
-
In vitro aging of β-amyloid protein causes peptide aggregation and neurotoxicity
-
Pike C. J., Walencewicz A. J., Glabe C. G., and Cotman C. W. (1991) In vitro aging of β-amyloid protein causes peptide aggregation and neurotoxicity. Brain Res. 563, 311-314.
-
(1991)
Brain Res.
, vol.563
, pp. 311-314
-
-
Pike, C.J.1
Walencewicz, A.J.2
Glabe, C.G.3
Cotman, C.W.4
-
25
-
-
0027447286
-
Neurodegeneration induced by β-amyloid peptides in vitro: The role of peptide assembly state
-
Pike C. J., Burdick D., Walencewicz A. J., Glabe C. G., and Cotman C. W. (1993) Neurodegeneration induced by β-amyloid peptides in vitro: the role of peptide assembly state. J. Neurosci. 13, 1676-1687.
-
(1993)
J. Neurosci.
, vol.13
, pp. 1676-1687
-
-
Pike, C.J.1
Burdick, D.2
Walencewicz, A.J.3
Glabe, C.G.4
Cotman, C.W.5
-
26
-
-
0028981219
-
Structure-activity analyses of β-amyloid peptides: Contributions of the β25-35 region to aggregation and neurotoxicity
-
Pike C. J., Walencewicz-Wasserman A. J., Kosmoski J., Cribbs D. H., Glabe C. G., and Cotman C. W. (1995) Structure-activity analyses of β-amyloid peptides: contributions of the β25-35 region to aggregation and neurotoxicity. J. Neurochem. 64, 253-265.
-
(1995)
J. Neurochem.
, vol.64
, pp. 253-265
-
-
Pike, C.J.1
Walencewicz-Wasserman, A.J.2
Kosmoski, J.3
Cribbs, D.H.4
Glabe, C.G.5
Cotman, C.W.6
-
27
-
-
85088330783
-
2-], homeostasis and induces neurotoxicity in cerebellar granule cells
-
2-], homeostasis and induces neurotoxicity in cerebellar granule cells. Soc. Neurosci. Abstr. 20, 602.
-
(1994)
Soc. Neurosci. Abstr.
, vol.20
, pp. 602
-
-
Scorziello, A.1
Meucci, O.2
Avallone, A.3
Galli, C.4
Schinelli, S.5
Salmona, M.6
Forloni, G.7
Schettini, G.8
-
28
-
-
0025753852
-
The molecular pathology of Alzheimer's disease
-
Selkoe D. J. (1991) The molecular pathology of Alzheimer's disease. Neuron 6, 487-498.
-
(1991)
Neuron
, vol.6
, pp. 487-498
-
-
Selkoe, D.J.1
-
29
-
-
0026646605
-
Isolation and quantitation of soluble Alzheimer's β-peptide from biological fluids
-
Seubert P., Vigo-Pelfrey C., Esch F., Lee M., Dovey H., Davis D., Sinha S., Schlossmacher M., Whaley J., Swindlehurst C., McCormack R., Wolfert R., Selkoe D. J., Lieberburg I., and Schenk D. (1992) Isolation and quantitation of soluble Alzheimer's β-peptide from biological fluids. Nature 359, 325-327.
-
(1992)
Nature
, vol.359
, pp. 325-327
-
-
Seubert, P.1
Vigo-Pelfrey, C.2
Esch, F.3
Lee, M.4
Dovey, H.5
Davis, D.6
Sinha, S.7
Schlossmacher, M.8
Whaley, J.9
Swindlehurst, C.10
McCormack, R.11
Wolfert, R.12
Selkoe, D.J.13
Lieberburg, I.14
Schenk, D.15
-
30
-
-
0027166738
-
Amyloid β peptides act directly on single neurons
-
Simmons M. A. and Schnaider C. R. (1993) Amyloid β peptides act directly on single neurons. Neurosci. Lett. 150, 133-136.
-
(1993)
Neurosci. Lett.
, vol.150
, pp. 133-136
-
-
Simmons, M.A.1
Schnaider, C.R.2
-
31
-
-
0022263028
-
The role of estrogens on the proliferation of human breast tumor cells (MCF-7)
-
Soto A. M. and Sonnenschein C. (1985) The role of estrogens on the proliferation of human breast tumor cells (MCF-7). J. Steroid Biochem. 23, 87-94.
-
(1985)
J. Steroid Biochem.
, vol.23
, pp. 87-94
-
-
Soto, A.M.1
Sonnenschein, C.2
-
32
-
-
0025832009
-
Membrane depolarization and the expression of glutamate receptors in cerebellar granule cells
-
Van der Valk J. B. F., Resink A., and Balazs R. (1991) Membrane depolarization and the expression of glutamate receptors in cerebellar granule cells. Eur. J. Pharmacol. 201, 247-250.
-
(1991)
Eur. J. Pharmacol.
, vol.201
, pp. 247-250
-
-
Van Der Valk, J.B.F.1
Resink, A.2
Balazs, R.3
-
33
-
-
0028180304
-
Ca2+ channel blockers attenuate β-amyloid peptide toxicity to cortical neurons in culture
-
Weiss J. H., Pike C. J., and Cotman C. W. (1994) Ca2+ channel blockers attenuate β-amyloid peptide toxicity to cortical neurons in culture. J. Neurochem. 62, 372-375.
-
(1994)
J. Neurochem.
, vol.62
, pp. 372-375
-
-
Weiss, J.H.1
Pike, C.J.2
Cotman, C.W.3
-
34
-
-
0024990330
-
Neurotrophic and neurotoxic effects of amyloid β-protein: Reversal by tachykinin neuropeptides
-
Yankner B. A., Duffy L. K., and Kirschner D. A. (1990) Neurotrophic and neurotoxic effects of amyloid β-protein: reversal by tachykinin neuropeptides. Science 250, 279-282.
-
(1990)
Science
, vol.250
, pp. 279-282
-
-
Yankner, B.A.1
Duffy, L.K.2
Kirschner, D.A.3
|