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Volumn 23, Issue 4, 1996, Pages 723-729

Aceclofenac, a new nonsteroidal antiinflammatory drug, decreases the expression and function of some adhesion molecules on human neutrophils

Author keywords

Aceclofenac; Adhesion molecules; L selectin; Neutrophil; NSAID

Indexed keywords

ACECLOFENAC; CD11B ANTIGEN; CD31 ANTIGEN; DEXAMETHASONE; DICLOFENAC; HERMES ANTIGEN; INDOMETACIN; INTERCELLULAR ADHESION MOLECULE 3; L SELECTIN; LEUKOSIALIN; LYMPHOCYTE FUNCTION ASSOCIATED ANTIGEN 1;

EID: 0029875122     PISSN: 0315162X     EISSN: None     Source Type: Journal    
DOI: None     Document Type: Article
Times cited : (43)

References (9)
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  • 4
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    • Nonsteroidal antiinflammatory drugs. Differences and similarities
    • Brooks PM, Day RO: Nonsteroidal antiinflammatory drugs. Differences and similarities. New Engl J Med 1991;24:1716-25.
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    • Brooks, P.M.1    Day, R.O.2
  • 5
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    • Neutrophil influx into an inflammatory site inhibited by a soluble horning receptor-IgG chimaera
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    • Watson, J.R.1    Fennie, C.2    Lasky, L.A.3
  • 8
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    • Enhancement of T cell activation by the CD43 molecule whose expression is defective in Wiskott-Aldrich syndrome
    • Park JK, Rosenstein Y, Remold-O'Donell E, Bierer BE, Rosen FS, Burakoff SI: Enhancement of T cell activation by the CD43 molecule whose expression is defective in Wiskott-Aldrich syndrome. Nature 1991;350:706-9.
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    • Park, J.K.1    Rosenstein, Y.2    Remold-O'Donell, E.3    Bierer, B.E.4    Rosen, F.S.5    Burakoff, S.I.6
  • 9
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    • The modulation of integrin-mediated T cell adhesion by CD43 is associated with its redistribution to a cellular uropod
    • Sánchez-Mateos P, Campanero MR, del Pozo MA, Sánchez-Madrid F: The modulation of integrin-mediated T cell adhesion by CD43 is associated with its redistribution to a cellular uropod. Blood 1995;86:2228-39.
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    • Sánchez-Mateos, P.1    Campanero, M.R.2    Del Pozo, M.A.3    Sánchez-Madrid, F.4


* 이 정보는 Elsevier사의 SCOPUS DB에서 KISTI가 분석하여 추출한 것입니다.