Tail proteins in cerebrospinal fluid from patients with alzheimer’s disease: A longitudinal study

Dementia and Geriatric Cognitive Disorders

Volumn 7, Issue 3, 1996, Pages 175-176

  Isoe, Kenji ^a   Urakami, Katsuya ^a   Shimomura, Tokio ^a   Wakutani, Yosuke ^a   Ji, Yong ^a   Adachi, Yoshiki ^a   Takahashi, Kazuro ^a  

^a TOTTORI UNIVERSITY   (Japan)

Author keywords

[No Author keywords available]

Indexed keywords

TAU PROTEIN;

ADULT; AGED; ALZHEIMER DISEASE; CEREBROSPINAL FLUID; CLINICAL ARTICLE; CLINICAL TRIAL; DIAGNOSTIC VALUE; DISEASE COURSE; DISEASE SEVERITY; FEMALE; HUMAN; LETTER; LONGITUDINAL STUDY; MALE; NERVE CELL LESION; NERVE CELL NECROSIS; PRIORITY JOURNAL;

AGED; AGED, 80 AND OVER; ALZHEIMER DISEASE; FEMALE; HUMANS; LONGITUDINAL STUDIES; MALE; MIDDLE AGED; TAU PROTEINS;

EID: 0029865711     PISSN: 14208008     EISSN: 14219824     Source Type: Journal    
DOI: 10.1159/000106874     Document Type: Article

References (5)

1. Vandermeeren M, Mercken M, Vanmeclielen E, Six J, Van de Voordc A, Martin JJ, Cras P: Detection of t proteins in normal and Alzheimer's disease cerebrospinal fluid with a sensitive sandwich enzyme-linked immunosorbent assay. J Neurochem 1993;61:1828-1834.
2. Mori H, Hosoda K, Matsubara E, Nakamoto T, Furiya Y, Endoh R, Usami M, Shoji M, Maruyama S, Hirai S: Tau in cerebrospinal fluids: Establishment of the sandwich ELISA with antibody specific to the repeat sequence in tau. Ncurosci Lett 1995:186:181-183.
3. Hock C, Golombowski S, Nascr W, Müller-Spahn F: Increased levels of t protein in cerebrospinal fluid of the patients with Alzheimer’s disease - correlation with degree of cognitive impairment. Ann Neurol 1995;37:414-415.
4. Urakami K, Takahashi K, Okada A, Oshima T, Adachi Y, Nakamura S, Kitaguchi N, Tokushima Y, Yamamoto S, Tanaka S: Clinical course and CSF amyloid β protein precursor having the site of application of the protcasc inhibitor (APPI) levels in patients with dementia of the Alzheimer type. Dementia 1993;4:59-60.
5. Mann DMA, Esiri MM: The site of the earliest lesions of Alzheimer’s disease. N Engl J Med 1988;318:789-790.