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Volumn 17, Issue 4, 1996, Pages 653-659

Diffuse plaques contain C-terminal Aβ42 and not Aβ40: Evidence from cats and dogs

Author keywords

Alzheimer's disease; Amyloid; Amyloid angiopathy; Animal models of aging; C terminal epitope; Canine; Feline; Senile plaque development

Indexed keywords

AMYLOID BETA PROTEIN;

EID: 0029835167     PISSN: 01974580     EISSN: None     Source Type: Journal    
DOI: 10.1016/0197-4580(96)00062-0     Document Type: Article
Times cited : (132)

References (9)
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  • 2
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    • Abnormally phosphorylated tau protein related to the formation of neurofibrillary tangles and neuropil threads in the cerebral cortex of sheep and goat
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    • Cataldo, A.M.1    Barnett, J.L.2    Berman, S.A.3    Li, J.4    Quarless, S.5    Bursztajn, S.6    Lippa, C.7    Nixon, R.A.8
  • 5
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    • The role of misdirected plasticity in plaque biogenesis and Alzheimer's disease pathology
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    • Cotman, C. W.; Cummings, B. J.; Whitson, J. S. The role of misdirected plasticity in plaque biogenesis and Alzheimer's disease pathology. In: Hefti, F.; Brachet, P.; Will, B.; Christen, Y., eds. Growth factors and Alzheimer's disease. Berlin: Springer Verlag; 1991:222-233.
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    • Cotman, C.W.1    Cummings, B.J.2    Whitson, J.S.3
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    • Image analysis of β-amyloid "load" in Alzheimer's disease and relation to dementia severity
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* 이 정보는 Elsevier사의 SCOPUS DB에서 KISTI가 분석하여 추출한 것입니다.