RXRγ null mice are apparently normal and compound RXRα+/-/RXRβ-/-/RXRγ-/- mutant mice are viable

Proceedings of the National Academy of Sciences of the United States of America

Volumn 93, Issue 17, 1996, Pages 9010-9014

  Krezel, Wojciech ^a   Dupé, Valerie ^a   Mark, Manuel ^a   Dierich, Andrée ^a   Kastner, Philippe ^a   Chambon, Pierre ^a  

^a PSL Research University   (France)

Author keywords

Knock out; Redundancy; Retinoid X receptor

Indexed keywords

RETINOID X RECEPTOR;

ANIMAL CELL; ANIMAL TISSUE; ARTICLE; CELL VIABILITY; DNA BINDING; GENE EXPRESSION; GENE MUTATION; HOMEOSTASIS; MOLECULAR CLONING; MOUSE; MUSCLE DEVELOPMENT; NONHUMAN; PHENOTYPE; PRIORITY JOURNAL; RETINOL DEFICIENCY; RNA ANALYSIS; SIGNAL TRANSDUCTION; SOUTHERN BLOTTING;

ANIMALS; BODY WEIGHT; HETEROZYGOTE; HOMOZYGOTE; MICE; MICE, KNOCKOUT; MOLECULAR SEQUENCE DATA; MUSCLE DEVELOPMENT; MUSCLE, SKELETAL; RECEPTORS, RETINOIC ACID; RETINOID X RECEPTORS; RNA, MESSENGER; TISSUE DISTRIBUTION; TRANSCRIPTION FACTORS;

ANIMALIA;

EID: 0029832219     PISSN: 00278424     EISSN: None     Source Type: Journal    
DOI: 10.1073/pnas.93.17.9010     Document Type: Article

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