Amiodarone-induced hypothyroidism followed by hyperthyroidism. In connection with two new observations;Hypothyroidie suivie d'une hyperthyroidie et induite par l'amiodarone. A propos de deux nouvelles observations

Annales de Medecine Interne

Volumn 147, Issue 5, 1996, Pages 369-372

  Wilhelm, Jean Marie ^a   Saraceni, Orlando ^a   Kieffer, Pierre ^a  

^a Centre Hospitalier Saint Morand   (France)

Author keywords

adverse effects; amiodarone; hyperthyroidism; hypothyroidism

Indexed keywords

AMIODARONE; LIOTHYRONINE; THYROTROPIN; THYROXINE; ANTIARRHYTHMIC AGENT;

ADULT; AGED; ARTICLE; CASE REPORT; FEMALE; HUMAN; HYPERTHYROIDISM; HYPOTHYROIDISM; MALE; PATHOPHYSIOLOGY; CHEMICALLY INDUCED DISORDER; MIDDLE AGED; REVIEW;

AGED; AMIODARONE; ANTI-ARRHYTHMIA AGENTS; FEMALE; HUMANS; HYPERTHYROIDISM; HYPOTHYROIDISM; MALE; MIDDLE AGED;

EID: 0029830871     PISSN: 0003410X     EISSN: None     Source Type: Journal    
DOI: None     Document Type: Article

References (20)

1. MASSIN JP, THOMOPOULOS P, KARAM J, SAVOIE JC : Le risque thyroïdien d'un nouveau coronarodilatateur iodé : l'amiodarone (Cordarone). Ann Endocrinol (Paris), 1971, 32 : 438-48.
2. HESHMATI HM, IZEMBART M, DEVAUX JP, VALLER G : Anticorps antirécepteurs de l'hormone thyréotrope dans l'hypothyroïdie auto-immune. Presse Med, 1987, 16 : 1376.
3. TAMAI J, KASAGI K, TAKAICHI Y et al. : Development of spontaneous hypothyroidism in patients with Grave's disease treated with antithyroidal drugs : clinical, immunological and histological findigs in 26 patients. J Clin Endocrinol Metab, 1989, 69 : 49-53.
4. LÉGER AF, MASSIN JP, LAURENT MF, VINCENS M et al. : Iodine-induced thyrotoxicosis : analysis of 85 consecutive cases. Eur J Clin Invest, 1984, 14 : 449-55.
5. SAVOIE JC, MASSIN JP, THOMOPOULOS P et al. : Iodine-induced thyrotoxicosis in apparently normal thyroid glands. J clin Endocrinol Metab, 1975, 41 : 685-91.
6. ROTI E, MINELLI R, GARDINI E et al. : Thyrotoxicosis followed by hypothyroidism in patients treated with amiodarone. Arch Intern Med, 1993, 153 : 886-92.
7. PATTE D, LÉGER FA, SAVOIE JC et al. : Thyreotoxicose, puis hypothyroïdie par surcharge iodée (amiodarone) associées à une neuropathie. Ann Med Interne (Paris), 1983, 134 : 31-4.
8. BRAMI JL, SCHLAEFFER F, SOBEL R : Amiodarone-induced dysthyroidism. Isr J Med Sci, 1985, 21 : 610-2.
9. KAPLAN J, ISH-SHAHOM S : Goiter and hypothyroidism during re-treatment with amiodarone in a patient who previously experienced amiodarone-induced thyrotoxicosis. Am J Med, 1991, 90 : 750-2.
10. ROTI E, MINELLA R, GARDINI E et al. : Iodine-induced subclinical hypothyroidism in euthyroid subject with a previous episode of amiodarone-induced thyrotoxicosis. J Clin Endocrinol Metab, 1992, 75 : 1273-7.
11. FATOURECHI V, GHARIB M : Hyperthyroidism following hypothyroidism. Arch Intern Med, 1988, 148 : 976-8.
12. TISON E, DEGROOTE P, WEMEAU JL et al. : Hypothyroïdie suivie d'une hyperthyroïdie. Sem Hop Paris, 1990, 66 (n° 16) : 841-3.
13. MINELLI R, GARDINI E, BIANCONI L et al. : Subclinical hypothyroidism, overt thyrotoxicosis and subclinical hypothyroidism : the subsequent phases of thyroid function in a patient chronically treated with amiodarone. J Endocrinol Invest, 1992, 15 : 853-5.
14. MARTIN-DU PAN PC, GOUMAZ MO, PERROTER C et al. : Hypothyroïdie suivie d'une hyperthyroïdie lors d'un traitement par l'amiodarone. Schweiz Med Wochenschr, 1993, 123 : 2110-2.
15. TRIP M, WIERSINGA W, THEO A, PLOMB TA : Incidence, predictability and pathogenesis of amiodarone-induced thyrotoxicosis and hypothyroidism. Am J Med, 1991, 91 : 507-11.
16. SALOMON E, ROWE RC, FAINAN C : Amiodarone-related cyclic thyroid dysfunction. CMAJ, 1989, 141 : 1247-9.
17. UNGER J, LAMBERT M, JONCKHEER MH, DENAYER P : Amiodarone and the thyroid : pharmacological, toxic and therapeutic effects. J Intern Med, 1993, 233 : 436-43
18. LAMBERT M, BRUGER A : Amiodarone and TRH stimulation test. Ana Clin Belg, 1982, 37 : 250-1.
19. JONCKHEER MH : Amiodarone and the thyroid gland. A review. Acta Cardiol, 1981, 36 : 199-205.
20. PARADIS P, LAMBERT C, ROULEAU J : Amiodarone antagonizes the effects of T3 at the receptor level ; an additional mechanism for its in vivo hypothyroid-like effects. Can J Physiol Pharmacol, 1991, 69 : 865-70.