Murine myeloid leukemic cells with disrupted myb loci show splicing anomalies that account for heterogeneous sizes in myb proteins

Oncogene

Volumn 13, Issue 6, 1996, Pages 1187-1196

  Tantravahi, Ramana ^a   Dudek, Henryk ^a   Patel, Geeta ^a   Reddy, E Premkumar ^a  

^a TEMPLE UNIVERSITY SCHOOL OF MEDICINE   (United States)

Author keywords

ABPL tumors; Myb protein heterogeneity; RNA splicing

Indexed keywords

COMPLEMENTARY DNA; GAG PROTEIN; HYBRID PROTEIN; MESSENGER RNA; ONCOPROTEIN; RIBONUCLEASE;

ANIMAL CELL; ARTICLE; CONTROLLED STUDY; DELETION MUTANT; ENVELOPE GENE; EXON; GENETIC HETEROGENEITY; IMMUNOPRECIPITATION; MOUSE; MUTAGENESIS; MYELOID LEUKEMIA; NONHUMAN; ONCOGENE C MYB; PRIORITY JOURNAL; PROTO ONCOGENE; RNA SPLICING; VIRUS CARCINOGENESIS;

AMINO ACID SEQUENCE; BASE SEQUENCE; CHROMOSOME MAPPING; GENE REARRANGEMENT; HUMANS; LEUKEMIA, MYELOID; MOLECULAR SEQUENCE DATA; POLYMERASE CHAIN REACTION; PROTEIN BIOSYNTHESIS; PROTO-ONCOGENE PROTEINS; PROTO-ONCOGENE PROTEINS C-MYB; RIBONUCLEASES; RNA SPLICING; RNA, MESSENGER; TRANS-ACTIVATION (GENETICS); TRANS-ACTIVATORS; TRANSCRIPTION, GENETIC; TRANSFORMATION, GENETIC; TUMOR CELLS, CULTURED;

ANIMALIA; MURINAE;

EID: 0029827052     PISSN: 09509232     EISSN: None     Source Type: Journal    
DOI: None     Document Type: Article

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