Aprotinin protects against myocardial and oxidant formation and endothelial cell damage during open heart surgery

Annals of the New York Academy of Sciences

Volumn 793, Issue , 1996, Pages 514-516

  Rao, Parinam S ^a   Palazzo, Robert S ^a   Bocchieri, Karl A ^a   Harlow, Greg L ^a   Metz, Helene N ^a   Wilson, David W ^a   Rao, Srinivasa K ^a   Graver, L Michael ^a  

^a ALBERT EINSTEIN COLLEGE OF MEDICINE   (United States)

Author keywords

[No Author keywords available]

Indexed keywords

APROTININ; GLUTATHIONE; HEMOGLOBIN; OXIDIZING AGENT;

CELL DAMAGE; CLINICAL ARTICLE; CLINICAL TRIAL; CONFERENCE PAPER; CONTROLLED CLINICAL TRIAL; CONTROLLED STUDY; CORONARY ARTERY BYPASS GRAFT; DRUG MECHANISM; ENDOTHELIUM CELL; HEART INFARCTION; HUMAN; OPEN HEART SURGERY; OXIDATION REDUCTION POTENTIAL;

EID: 0029804136     PISSN: 00778923     EISSN: None     Source Type: Book Series    
DOI: 10.1111/j.1749-6632.1996.tb33553.x     Document Type: Conference Paper

References (3)

1. ALVEREZ, J. et al. 1995. The use of ultra low dose aprotinin to reduce blood loss in cardiac surgery. J. Cardiotho. Vascu. Anasthe. 9(1): 29-33.
2. ZANNIONI, V., M. LYNCH, S. GOLDSTEIN & P. SATO. 1974. A rapid micro method for determination of ascorbic acid in plasma and tissues. Biochem. Med. 11: 41-44.
3. RAO, P. S., G. S. WEINSTEIN, N. RUJIKARA, J. M. LUBER & D. H. TYRAS. 1990. An HPLC method for in vivo quantitation of oxygen free radicals using spin and chemical traps. Chromatographia 30: 19-23.