Transforming growth factor β1 suppresses genomic instability independent of a G1 arrest, p53, and Rb

Cancer Research

Volumn 56, Issue 16, 1996, Pages 3645-3650

  Glick, Adam B ^a   Weinberg, Wendy C ^a   Wu, I Hui ^a   Quan, Wing ^a   Yuspa, Stuart H ^a  

^a NATIONAL CANCER INSTITUTE   (United States)

Author keywords

[No Author keywords available]

Indexed keywords

PROTEIN P53; RECEPTOR SUBTYPE; RETINOBLASTOMA PROTEIN; SPARFOSIC ACID; TRANSFORMING GROWTH FACTOR BETA RECEPTOR; TRANSFORMING GROWTH FACTOR BETA1;

ANIMAL CELL; ARTICLE; CELL CYCLE G1 PHASE; CELL LINE; CELL PROLIFERATION; CONTROLLED STUDY; GENE AMPLIFICATION; GENE DELETION; GENETIC STABILITY; GENOTYPE; GROWTH INHIBITION; HUMAN; HUMAN CELL; KERATINOCYTE; MOUSE; NEWBORN; NONHUMAN; PRIORITY JOURNAL; SIGNAL TRANSDUCTION; TUMOR CELL LINE; TUMOR GROWTH;

ANIMALS; ASPARTIC ACID; CELLS, CULTURED; G1 PHASE; GENE AMPLIFICATION; GENES, P53; GENES, RETINOBLASTOMA; MICE; PHOSPHONOACETIC ACID; TRANSFORMING GROWTH FACTOR BETA;

EID: 0029784067     PISSN: 00085472     EISSN: None     Source Type: Journal    
DOI: None     Document Type: Article

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