Neuropathology and pathogenesis of mitochondrial diseases

Journal of Inherited Metabolic Disease

Volumn 19, Issue 4, 1996, Pages 553-572

  Brown, G K ^a   Squier, M V ^b  

^a UNIVERSITY OF OXFORD   (United Kingdom)

^b RADCLIFFE INFIRMARY   (United Kingdom)

Author keywords

[No Author keywords available]

Indexed keywords

ADENOSINE TRIPHOSPHATE; EXCITATORY AMINO ACID; ION CHANNEL; LACTIC ACID; NEUROTRANSMITTER; NITRIC OXIDE; OXYGEN RADICAL;

ASTROCYTE; BRAIN HYPOXIA; BRAIN ISCHEMIA; BRAIN METABOLISM; BRAIN SPONGIOSIS; CLINICAL FEATURE; CONFERENCE PAPER; DISEASE COURSE; DISEASE SEVERITY; DISORDERS OF MITOCHONDRIAL FUNCTIONS; ELECTRIC ACTIVITY; ENERGY METABOLISM; GLIOSIS; HUMAN; HYPOGLYCEMIA; LEIGH DISEASE; NERVE CELL NECROSIS; NEUROPATHOLOGY; PATHOGENESIS; RESPIRATORY CHAIN;

EID: 0029763179     PISSN: 01418955     EISSN: None     Source Type: Journal    
DOI: 10.1007/BF01799116     Document Type: Conference Paper

References (3)

1. Adornato B, Lamport P (1971) Status spongiosus of nervous tissue: electron microscopic studies. Acta Neuropathol 19: 271-289.
2. Albin RL, Greenamyre JT (1992) Alternative excitotoxic hypotheses. Neurology 42: 733-738.
3. Ashcroft FM (1988) Adenosine 5′-triphosphate-sensitive potassium channels. Annu Rev Neurosci 11: 97-118.