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Odawara T, Iseki E, Kosaka K, Haga C, Ikeda K: Incidence and distribution of senile plaques and neurofibrillary tangles in late-onset diffuse Lewy body disease. Neuropathology 1994, 14:7-11.
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Lippa CF, Smith TW, Nee L, Robitaille Y, Crain B, Dickson D, Pulaski Salo D, Pollen DA: Familial Alzheimer's disease and cortical Lewy bodies: is there a genetic susceptibility factor? Dementia 1995, 6:191-194.
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Lippa, C.F.1
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17
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0028907906
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'Lewy body disease': Clinico-pathological correlation in 18 consecutive cases of Parkinson's disease with and without dementia
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de Vos RAI, Jansen ENH, Stam FC, Ravid R, Swaab DF: 'Lewy body disease': clinico-pathological correlation in 18 consecutive cases of Parkinson's disease with and without dementia. Clin Neurol Neurosurg 1995, 97:13-22.
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Wakabayashi K, Hansen LA, Masliah E: Cortical Lewy body-containing neurons are pyramidal cells: imaging of double-immunolabeled sections with anti-ubiquitin and SMI32. Acta Neuropathol 1995, 89:404-408. The authors showed by using an immunostaining method that cortical Lewy body-containing neurons are pyramidal cells.
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Cu/Zn Superoxide dismutase-like immunoreactivity is present in Lewy bodies from Parkinson disease: A light and electron microscopic immunocytochemical study
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Nishiyama K, Murayama S, Shimizu J, Ohya Y, Kwak S, Asayama K, Kanazawa I: Cu/Zn Superoxide dismutase-like immunoreactivity is present in Lewy bodies from Parkinson disease: a light and electron microscopic immunocytochemical study. Acta Neuropathol 1995, 89:471-474. Lewy bodies in Parkinson's disease were shown to be immunostained with an antibody against Cu/Zn Superoxide dismutase.
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Nishiyama, K.1
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Hippocampal degeneration differentiates diffuse Lewy body disease (DLBD) from Alzheimer's disease: Light and electron microscopic immunocytochemlstry of CA2-3 neurites specific to DLBD
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Dickson DW, Ruan D, Crystal H: Hippocampal degeneration differentiates diffuse Lewy body disease (DLBD) from Alzheimer's disease: light and electron microscopic immunocytochemlstry of CA2-3 neurites specific to DLBD. Neurology 1991, 41:1402-1409.
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Kim H, Gearing M, Mirra SS: Ubiquitin-positive CA2/3 neurites in hippocampus coexist with cortical Lewy bodies. Neurology 1995, 45:1768-1770.
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A pathological study of Lewy bodies and senile changes in the amygdala in diffuse Lewy body disease
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Iseki E, Odawara T, Suzuki K, Kosaka K, Akiyama H, Ikeda K: A pathological study of Lewy bodies and senile changes in the amygdala in diffuse Lewy body disease. Neuropathology 1995, 15:112-116. The authors showed in detail the distribution and incidence of Lewy bodies and senile changes in amygdala in DLBD patients.
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Smith, T.W.1
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Investigation of tau-2 positive microglia-like cells in the subcortical nuclei of human neurodegenerative disorders
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Odawara T, Iseki E, Kosaka K, Akiyama H, Ikeda K, Yamamoto T: Investigation of tau-2 positive microglia-like cells in the subcortical nuclei of human neurodegenerative disorders. Neurosci Lett 1995, 192:145-148.
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Williams, T.L.1
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Hedera, P.1
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Kosaka, K.1
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Alteration in nicotine binding sites in Parkinson's disease, Lewy body dementia and Alzheimer's disease: Possible index of early neuropathology
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Perry EK, Morris CM, Court JA, Cheng A, Fainbairn AF, McKeith IG, Irving P, Brown A, Perry RH: Alteration in nicotine binding sites in Parkinson's disease, Lewy body dementia and Alzheimer's disease: possible index of early neuropathology. Neuroscience 1995, 64:385-395. High-affinity nicotine binding was examined autoradiographically in brain regions most severely affected in ATD, DLB, and Parkinson's disease. The authors pointed out that loss or downregulation of the receptor may precede neurodegeneration.
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Comparison of cathepsin protease activities in brain tissue from normal cases and with Alzheimer's disease, Lewy body dementia, Parkinson's disease and Huntington's disease
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Mantle D, Falkous G, Ishiura S, Perry RH, Perry EK: Comparison of cathepsin protease activities in brain tissue from normal cases and with Alzheimer's disease, Lewy body dementia, Parkinson's disease and Huntington's disease. J Neurol Sci 1995, 131:65-70.
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Mantle, D.1
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Scot HL, Tannenberg AE, Dodd PR: Variant forms of neuronal glutamate transporter sites in Alzheimer's disease cerebral cortex. J Neurochem 1995, 64:2193-2202.
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Scot, H.L.1
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Lippa CF, Smith TW, Saunders AM, Crook R, Pulaski Salo D, Davies P, Hardy J, Roses AD, Dickson D: Apolipoprotein E genotype and Lewy body disease. Neurology 1995, 45:97-103. The relation between apoE genotype and neuropathology was studied in patients with DLB. The authors suggest that apoE genotype may affect neuropathology in DLB but that it may not have a major direct effect on Lewy body formation.
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Lippa, C.F.1
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The CYP2D6B mutant allele is overrepresented in the Lewy body variant of Alzheimer's disease
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Saitoh T, Xia Y, Chen X, Masliah E, Galasko D, Shults G, Thai LJ, Hansen LA, Katzman R: The CYP2D6B mutant allele is overrepresented in the Lewy body variant of Alzheimer's disease. Ann Neurol 1995, 37:110-112. The authors reported a higher representation of the cytochrome P450 CPY2D6 mutant B allele in LBV than in ATD and suggested that this allele is a risk factor for both Parkinson's disease and LBV but not for ATD.
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Saitoh, T.1
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Distribution of brain cytochrome oxidase activity in various neurodegenerative diseases
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de la Monte SM, Ng SC, Hsu DW: Aberrant GAP-43 gene expression in Alzheimer's disease. Am J Pathol 1995, 147:934-946.
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De La Monte, S.M.1
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Allen RL, Walker Z, O'Ath PJ, Katona CL: Risperidone for psychotic and behavioural symptoms in Lewy body dementia [Letter]. Lancet 1995, 346:185. The authors reported that low doses of risperidone produced useful anti-psychotic effects in three patients with DLB.
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Allen, R.L.1
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McKeith, I.G.1
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Neocortical cholinergic activities differentiate Lewy body dementia from classical Alzheimer's disease
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Perry EK, Haroutunian V, Davis KL, Lantos P, Eagger S, Honavar M, Dean A, Griffiths M, McKeith IG, Perry RH: Neocortical cholinergic activities differentiate Lewy body dementia from classical Alzheimer's disease. NeuroReport 1994, 5:747-749.
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Perry, E.K.1
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Levy R, Egger S, Griffiths M, Perry EK, Honavar M, Dean A, Lantos P: Lewy bodies and response to tacrine in Alzheimer's disease [Letter]. Lancet 1994, 343:176.
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Lancet
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Levy, R.1
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Liberini P, Memo M, Spano P: Lewy body pathology and heterogeneity of Alzheimer's disease [Letter]. JAMA 1995, 274:1199. The authors hypothesized that the subgroups of Alzheimer's disease patients who responded positively to tacrine are patients affected by DLB either with or without concomitant Alzheimer's disease.
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Liberini, P.1
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