Inactivation of hepatitis C virus cDNA transgene by hypermethylation in transgenic mice

Archives of Virology

Volumn 141, Issue 5, 1996, Pages 951-958

  Kato, T ^a  

^a NIHON UNIVERSITY SCHOOL OF MEDICINE   (Japan)

Author keywords

[No Author keywords available]

Indexed keywords

COMPLEMENTARY DNA; VIRUS DNA;

ANIMAL; ARTICLE; C57BL MOUSE; GENETICS; HEPATITIS C VIRUS; METABOLISM; METHYLATION; MOLECULAR GENETICS; MOUSE; NUCLEOTIDE SEQUENCE; TRANSGENE; TRANSGENIC MOUSE;

ANIMALS; BASE SEQUENCE; DNA, COMPLEMENTARY; DNA, VIRAL; HEPACIVIRUS; METHYLATION; MICE; MICE, INBRED C57BL; MICE, TRANSGENIC; MOLECULAR SEQUENCE DATA; TRANSGENES;

EID: 0029692895     PISSN: 03048608     EISSN: None     Source Type: Journal    
DOI: 10.1007/BF01718169     Document Type: Article

References (2)

1. Araki K, Miyazaki J, Tsurimoto T, Inomoto T, Iwanaga T, Matsubara K, and Yamamura K (1989) Demethylation by 5-azacytidine results in the expression of hepatitis B virus surface antigen in transgenic mice. Jpn J Cancer Res 80: 295-298
2. Araki K, Akagi K, Miyazaki J, Matsubara K, Yamamura K (1990) Correlation of tissue specific methylaton with gene inactivity in hepatitis B virus transgenic mice. Jpn J Cancer Res 81:1265-1271