A reference value for delta-aminolevulinic acid in plasma in the population occupationally unexposed to lead

Industrial Health

Volumn 34, Issue 1, 1996, Pages 57-60

  Morita, Yoko ^a   Sakai, Tadashi ^b   Araki, Takaharu ^b   Suzuki, Koichi ^c   Oda, Kenichi ^c   Araki, Shunichi ^d   Masuyama, Yoshiaki ^b  

^a NATL INSTITUTE OF INDUSTRIAL HEALTH   (Japan)

^b Center of Occupational Medicine   (Japan)

^c Derpartment of Health Examination   (Japan)

^d UNIVERSITY OF TOKYO   (Japan)

Author keywords

Delta aminolevulinic acid; Plasma; Reference value

Indexed keywords

AMINOLEVULINIC ACID; EDETIC ACID;

ADULT; ARTICLE; BLOOD SAMPLING; CONTROLLED STUDY; DATA ANALYSIS; FEMALE; HIGH PERFORMANCE LIQUID CHROMATOGRAPHY; HUMAN; HUMAN EXPERIMENT; LEAD BLOOD LEVEL; MALE; NORMAL HUMAN; OCCUPATIONAL EXPOSURE; PHYSICAL EXAMINATION; REFERENCE VALUE;

EID: 0029671370     PISSN: 00198366     EISSN: None     Source Type: Journal    
DOI: 10.2486/indhealth.34.57     Document Type: Article

References (12)

1. Muller WE, Synder SH. δ-Aminolevulinic acid: Influences on synaptic GABA receptor binding may explain CNS symptoms of porphyria. Ann Neurol 1977; 2: 340-2.
2. Thunell S, Holmberg L, Lundgen J. Aminolaevulinate dehydratase porphyria in infancy. A clinical and biochemical study. J Clin Chem Clin Biochem 1987; 25: 5-14.
3. Fraga CG, Onuki J, Lucesoli F, Bechara EJH, diMascio P. 5-aminolevulinic acid mediates the in vivo and in vitro formation of 8-hydroxy-2′-deoxyguanosine in DNA. Carcinogenesis 1994; 10: 2241-4
4. Hermes-Lima M, Valle VGR, Vercesi AE, Bechara EJH. Damage to rat liver mitochondria promoted by δ-aminolevulinic acid - generated reactive oxygen species. Connections with acute intermittent porphyria and lead-poisoning. Biochim Biophys Acta 1991; 1056: 57-63.
5. Hosoda K, Sakurai H, Omae K, Kamakura M, Satoh T. δ-aminolevulinic acid in blood as an indicator of early health effect of lead. Jpn J Ind Health 1989; 31: 240-1.
6. Takebayashi T, Omae K, Hosoda K, Satoh T, Hamaguchi T, Sakurai H. Evaluation of δ-aminolevulinic acid in blood of workers exposed to lead. Br J Ind Med 1993; 50: 49-5.
7. Tomokuni K, Ichiba M, Hirai Y. High-sensitive determination of δ-aminolevulinic acid (ALA) in serum or plasma. Porphyrins 1992; 1: 299-304.
8. Morita Y, Araki S, Sakai T, Araki T, Masuyama Y. Determination of delta-aminolevulinic acid in plasma using high-performance liquid chromatography: a sensitive indicator of lead effects. Ind Health 1994; 32: 85-96.
9. Kasahara A. Studies on urinary δ-aminolevulinic acid excretion of healthy urban male population. Osaka City Med J 1965; 11: 99-117.
10. Watanabe T, Fujita H, Koizumi A, Chiba K, Miyasaka M, Ikeda M. Baseline level of blood lead concentration among Japanese farmers. Arch Environ Health 1985; 40: 170-6.
11. Taguchi T, Suzuki S, Yokohashi G. Normal levels of lead, δ-aminolevulinic acid and coproporphyrin in spot urines. Jpn J Ind Health 1970; 12: 482-3.
12. Sakai T, Morita Y. S-Aminolevulinic acid in plasma or whole blood as a sensitive indicator of lead effects, and its relation to the heme-related parameters. Int Arch Occup Environ Health (in press).