Decreased expression of both FcγRII and FcγRIII mRNA in leukemic granulocytes

Leukemia Research

Volumn 19, Issue 12, 1995, Pages 997-1000

  Kant, Anita ^a   Advani, Suresh ^b   Zingde, Surekha ^a  

^a CANCER RESEARCH INSTITUTE   (India)

^b TATA MEMORIAL HOSPITAL   (India)

Author keywords

CML; Fc RII; Fc RIII; granulocyte; leukemia

Indexed keywords

FC RECEPTOR; IMMUNOGLOBULIN G; MESSENGER RNA;

ARTICLE; CHRONIC MYELOID LEUKEMIA; CONTROLLED STUDY; GENE EXPRESSION; GRANULOCYTE; HUMAN; HUMAN CELL; INTERNALIZATION; LEUKEMIA CELL; PRIORITY JOURNAL; RECEPTOR GENE;

BLOTTING, NORTHERN; GRANULOCYTES; HUMANS; LEUKEMIA, MYELOID, CHRONIC; RECEPTORS, IGG; RNA, MESSENGER;

EID: 0029417343     PISSN: 01452126     EISSN: None     Source Type: Journal    
DOI: 10.1016/0145-2126(95)00094-1     Document Type: Article

References (28)

1. 2, CD11b/CD18) and FcγRIII cooperate in generation of a neutrophil respiratory burst: requirement for FcγRII and tyrosine phosphorylation
2. Tyrosine phosphorylation induced by cross-linking of Fcγ-receptor type II in human neutrophils
3. Human low-affinity IgG receptor FcγRIIa (CD32) introduced into mouse fibroblasts mediates phagocytosis of sensitized erythrocytes
4. The 40 kDa Fcγ receptor (FcRII) on human neutrophils is essential for the IgG-induced respiratory burst and IgG-induced phagocytosis
5. Reconstitution of antibody-dependent phagocytosis in fibroblast expressing Fcγ receptor IIIB and the complement receptor type 3
6. Activatation of FcγRII induces tyrosine phosphorylation of multiple proteins including Fcγ RII
7. Biology of chronic myeloid leukemia: Is discordant maturation the primary defect?
8. The receptor for IgG on human normal and chronic myeloid leukemic granulocytes: functional and biochemical characterisation
9. Decreased expression of FcγRIII mRNA in leukemic granulocytes
10. Differential phosphorylation—cause for defective internalization of aggregated IgG by chronic myeloid leukemic granulocytes?
11. Separation of blood leukocytes, granulocytes and lymphocytes
12. Single step method of RNA isolation by acid guanidinium thiocyanate-phenol-chloroform extraction
13. Electrophoresis of RNA through gels containing formaldehyde
14. Isolation and expression of cDNA clones encoding a human receptor for IgG (FcγRII)
15. Human FcγRIII: cloning, expression, and identification of the chromosomal locus of two receptors for IgG
16. Isolation of plasmid DNA
17. Differential expression of FcγRII A, FcγRII B, FcγRII C in hematopoietic cells: analysis of transcripts
18. FcγRIII (CD16) expression on neutrophils from chronic myeloid leukemia, a flow cytometric study
19. FUNCTIONAL AND BIOCHEMICAL PHENOTYPE IN RELATION TO CELLULAR AGE OF DIFFERENTIATED NEUTROPHILS IN CHRONIC MYELOID LEUKAEMIA
20. Differences in the expression of alkaline phosphatase mRNA in chronic myeloid leukemia and paroxysmal nocturnal hemoglobinuria polymorphonuclear leukocytes
21. Deficiency of a phosphatidyl inositol-anchored cell adhesion molecule influences haemopoietic progenitor binding to marrow stroma in chronic myeloid leukemia
22. The major Fc receptor in blood has a phosphatidylinositol anchor and is deficient in paroxysmal nocturnal hemoglobinuria
23. Affected erythrocytes of patients with paroxysmal nocturnal hemoglobinuria are deficient in the complement regulatory protein, decay accelerating factor.
24. Neutrophil (leukocyte) alkaline phosphatase in paroxysmal nocturnal hemoglobinuria
25. Phosphatidylinositol-linked proteins and paroxysmal nocturnal hemoglobinuria
26. Specific defect in N-acetylglucosamine incorporation in the biosynthesis of the glycosylphosphatidylinositol anchor in cloned cell lines from patients with paroxysmal nocturnal hemoglobinuria
27. Deficient biosynthesis of N-acetylglucosaminyl-phosphatidylinositol, the first intermediate of glycosylphosphatidylinositol anchor biosynthesis, in cell lines established from patients with paroxysmal nocturnal hemoglobinuria