Hypersecretion of mucin in response to inflammatory mediators by guinea pig tracheal epithelial cells in vitro is blocked by inhibition of nitric oxide synthase.

American journal of respiratory cell and molecular biology

Volumn 13, Issue 5, 1995, Pages 526-530

  Adler, K B ^a   Fischer, B M ^a   Li, H ^a   Choe, N H ^a   Wright, D T ^a  

^a NORTH CAROLINA STATE UNIVERSITY   (United States)

Author keywords

[No Author keywords available]

Indexed keywords

ARGININE; AUTACOID; DRUG DERIVATIVE; ENZYME INHIBITOR; HISTAMINE; MUCIN; N(G) METHYLARGININE; NITRIC OXIDE SYNTHASE; SUPEROXIDE; THROMBOCYTE ACTIVATING FACTOR; TUMOR NECROSIS FACTOR ALPHA;

ANIMAL; ARTICLE; CELL CULTURE; DRUG ANTAGONISM; DRUG EFFECT; GUINEA PIG; METABOLISM; SECRETION; TRACHEA;

ANIMALS; ARGININE; CELLS, CULTURED; ENZYME INHIBITORS; GUINEA PIGS; HISTAMINE; INFLAMMATION MEDIATORS; MUCINS; NITRIC OXIDE SYNTHASE; OMEGA-N-METHYLARGININE; PLATELET ACTIVATING FACTOR; SECRETORY RATE; SUPEROXIDES; TRACHEA; TUMOR NECROSIS FACTOR-ALPHA;

EID: 0029410613     PISSN: 10441549     EISSN: None     Source Type: Journal    
DOI: 10.1165/ajrcmb.13.5.7576687     Document Type: Article

References (0)