Enhanced vulnerability to secondary ischemic insults after experimental traumatic brain injury

New Horizons: Science and Practice of Acute Medicine

Volumn 3, Issue 3, 1995, Pages 376-383

  DeWitt, D S ^a   Jenkins, L W ^a   Prough, D S ^a  

^a UNIVERSITY OF TEXAS MEDICAL BRANCH   (United States)

Author keywords

cerebral blood flow; cerebral metabolism; excitotoxicity; free radicals; glutamate; ischemic brain injury; protein kinase C; superoxide dismutase; traumatic brain injury

Indexed keywords

ARACHIDONIC ACID DERIVATIVE; CALCIUM ION; GLUTAMIC ACID; OXYGEN RADICAL; PROTEIN KINASE C; SUPEROXIDE DISMUTASE;

ANEMIA; BLOOD BRAIN BARRIER; BRAIN BLOOD FLOW; BRAIN INJURY; BRAIN ISCHEMIA; BRAIN METABOLISM; BRAIN PERFUSION; DISEASE PREDISPOSITION; HUMAN; HYPOTENSION; HYPOXEMIA; NONHUMAN; OXYGEN TRANSPORT; PERFUSION PRESSURE; PRIORITY JOURNAL; REVIEW;

ANIMAL; BRAIN INJURIES; BRAIN ISCHEMIA; CATS; CEREBROVASCULAR CIRCULATION; DISEASE SUSCEPTIBILITY; HUMAN; HYPOXIA, BRAIN; RATS; SUPPORT, U.S. GOV'T, P.H.S.;

EID: 0029155453     PISSN: 10637389     EISSN: None     Source Type: Journal    
DOI: None     Document Type: Review

References (0)