Different effects on gating of three myotonia‐causing mutations in the inactivation gate of the human muscle sodium channel.

The Journal of Physiology

Volumn 487, Issue 1, 1995, Pages 107-114

  Mitrovie N ^a   George, A L ^a   Lerche, H ^a   Wagner, S ^a   Fahlke, C ^a   Lehmann Horn F ^a  

^a UNIVERSITY OF ULM   (Germany)

Author keywords

[No Author keywords available]

Indexed keywords

POTASSIUM ION; SODIUM CHANNEL; SODIUM ION;

ARTICLE; CHANNEL GATING; CONTROLLED STUDY; DISEASE SEVERITY; ELECTRIC POTENTIAL; EMBRYO; GENE MUTATION; HUMAN; HUMAN CELL; KIDNEY CELL; MUSCLE; MYOTONIA; PHENOTYPE; PRIORITY JOURNAL; SODIUM CURRENT; TIME;

BASE SEQUENCE; CELL LINE; GENOTYPE; HUMAN; ION CHANNEL GATING; MOLECULAR SEQUENCE DATA; MUSCLES; MUTATION; MYOTONIA; OLIGONUCLEOTIDE PROBES; PHENOTYPE; POTASSIUM; RECOMBINANT PROTEINS; SODIUM CHANNELS; SUPPORT, NON-U.S. GOV'T; SUPPORT, U.S. GOV'T, P.H.S.;

EID: 0029131274     PISSN: 00223751     EISSN: 14697793     Source Type: Journal    
DOI: 10.1113/jphysiol.1995.sp020864     Document Type: Article

References (0)