A checkpoint regulates the rate of progression through S phase in S. cerevisiae in Response to DNA damage

Cell

Volumn 82, Issue 5, 1995, Pages 841-847

  Paulovich, Amanda G ^a   Hartwell, Leland H ^a  

^a UNIVERSITY OF WASHINGTON   (United States)

Author keywords

[No Author keywords available]

Indexed keywords

DNA; GENE PRODUCT;

ARTICLE; ATAXIA TELANGIECTASIA; CELL CYCLE S PHASE; DNA DAMAGE; DNA SYNTHESIS; GENE MUTATION; GENE REPLICATION; MITOSIS INHIBITION; NONHUMAN; PRIORITY JOURNAL; SACCHAROMYCES CEREVISIAE; SEQUENCE HOMOLOGY;

ATAXIA; ATAXIA TELANGIECTASIA; MAMMALIA; SACCHAROMYCES CEREVISIAE;

EID: 0029085781     PISSN: 00928674     EISSN: None     Source Type: Journal    
DOI: 10.1016/0092-8674(95)90481-6     Document Type: Article

References (39)

1. 3-Methyladenine residues in DNA induce the SOS function sfiA in Escherichia coli
2. The relevance of the nuclear division cycle to radiosensitivity in yeast
3. Cell cycle specific repair of DNA double strand breaks in Saccharomyces cerevisiae
4. The action of caffeine on X-irradiated HeLa cells. III. Enhancement of X-ray-induced killing during G2 arrest
5. Fission yeast genes involved in coupling mitosis to completion of DNA replication
6. TEL1, a gene involved in controlling telomere length in S. cerevisiae, is homologous to the human ataxia telangiectasia gene
7. Checkpoints: controls that ensure the order of cell cycle events
8. Replication-dependent sister chromatid recombination in radl mutants of Saccharomyces cerevisiae
9. A mammalian cell cycle checkpoint pathway utilizing p53 and GADD45 is defective in ataxia-telangiectasia
10. Radiation effects on DNA synthesis in a defined chromosomal replicon
11. Strand-selective repair of DNA damage in the yeast GAL7 gene requires RNA polymerase II
12. TEL1 an S cerevisiae homolog of the gene mutated in ataxia telangiectasia is functionally related to the yeast checkpoint gene MEC1
13. Creative blocks: cell-cycle checkpoints and feedback controls
14. The genetics of cell cycle checkpoints
15. Roadblocks and detours during DNA replication: mechanisms of mutagenesis in mammalian cells
16. DNA polymerase s links the DNA replication machinery to the ϵ phase checkpoint
17. Radiosensitivity in ataxiatelangiectasia: a new explanation
18. Chromosome end associations, telomeres and telomerase activity in ataxia telangiectasia cells
19. Defective G2 checkpoint function in cells from individuals with familial cancer syndromes
20. Preferential synthesis of yeast mitochondrial DNA in a factor-arrested cells
21. The repair of double-strand breaks in the nuclear DNA of Saccharomyces cerevisiae and its genetic control
22. A single ataxia telangiectasia gene with a product similar to PI-3 kinase
23. Molecular mechanism of transcription-repair coupling
24. RAD9dependent G1 arrest defines a second checkpoint for damaged DNA in the cell cycle of Saccharomyces cerevisiae
25. Characterization of G1 checkpoint control in the yeast Saccharomyces cerevisiaefollowing exposureto DNA-damaging agents
26. Site-specific DNA repair at the nucleosomal level in a yeast minichromosome
27. Preferential repair of cyclobutane pyrimidine dimers in the transcribed strand of a gene in yeast chromosomes and plasmids is dependent on transcription
28. The incidence and gene frequency of ataxia-telangiectasia in the United States
29. Breast and other cancers in families with ataxia-telangiectasia
30. Incidence of cancer in 161 families affected by ataxia-telangiectasia
31. The double-strand-break repair model for recombination
32. The p21 inhibitor of cyclin-dependent kinases controls DNA replication by interaction with PCNA
33. The RAD9 gene controls the cell cycle response to DNA damage in Saccharomyces cerevisiae
34. Cell cycle arrest of cdc mutants and specificity of the RAD9 checkpoint
35. Mitotic checkpoint genes in budding yeast and the dependence of mitosis on DNA replication and repair
36. Radioresistant DNA synthesis and human genetic diseases
37. Cell death, chromosome damage and mitotic delay in normal human, ataxia telangiectasia and retinoblastoma fibroblasts after X-irradiation